Colon cancer a pathophysiology ngu ai gaw, colon cancer byin wa ai lam hte rawt jat wa ai lam hta lawm ai lam ni hpe tsun ai re.
Colon cancer ngu ai gaw, kan kata kaw nna hpang ai cancer rai nga ai.
Dai gaw, ana byin hpang ai shaloi, ana kap ai zawn byin wa nna, hpang de gaw, cancer byin wa chye ai.
Colon cancer a pathophysiology hta genetic mutations, inflammation, hte environmental factor ni lawm ai lam law law lawm ai.
Colon cell ni a DNA hta genetic mutation byin wa nna, n hkang lu ai hku cell ni kaba wa ai hte garan wa ai lam byin wa chye ai.
Dai galai shai ai lam ni gaw, sali wunli hte seng nna lu la ai (sh) lu la ai lam ni rai nna, dai gaw, hkum hkrang hta nga ai, hkum hkrang daw wa ai, bai gram lajang ai lam ni hta lawm ai, amyu baw hpan ni hpe hkra machyi shangun ai.
Nbawn hta ana byin wa ai lam gaw, nbawn ana zawn re ai ana ni a majaw byin wa chye ai.
Hkum hkrang hta ana byin ai shaloi, hkum hkrang hta nga ai hpa ni gaw, hkum hkrang hta nga ai kaga hkum hkrang ni hpe sawng wa hkra galaw ya ai.
Htingra masa lam ni, lu sha ai lam, sak hkrung lam hte kaga hpaga lailen ni gaw, pu kan ana byin wa hkra galaw ya lu ai.
Ga shadawn, shan hkyeng hte shan galaw sha ai hta jan ai gaw, kansi ana byin chye ai lam grau law wa ai.
Kaga lam ni mung, gawng kya ai, tsabyi lu ai, hkum hkrang n shamu ai lam ni gaw, dai ana byin wa hkra grau galaw ya ai.
Hkum shan hta kalang mi byin wa ai colon cancer gaw, hkum hkrang a kaga daw de mung chyam bra wa ai.
Dai cancer gaw makau grup yin na sai hte daw shan ni hpe shang wa nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai hte sai lam hku nna sai lam hku nna sai lam hku nna sai lam hku nna sai lam hku nna sai lam hku nna sai lam hku
Hkum shan hta byin ai cancer ana hpe tsi lajang ai lam hta, hkala sawng ai madang hte shara hta hkan nna, surgery, chemotherapy hte radiation therapy ni hpe jawm galaw ra ai.
Dai majaw, colon cancer hpe shawng nnan mu tam ai shaloi, mai tsi kau ai lam nga ai.
Colonoscopy zawn re ai chyahkring hkring sawk sagawn ai lam ni gaw, ana hpe atsawm sha tsi mai ai aten hta, shawng nnan hta ana hpe mu tam lu ai.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
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What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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