Colon cancer, shiro colorectal cancer lan notǝna dǝ, shima jili kansa do suro kǝmbaram colon au rectum lan baditin ma, sandima nasha la suro kǝmbaram kura ye dǝn.
Kambosoro awo kura laa shiro polyp gultin lan baditin, shi doni letǝram lan kansa ro waljin ma.
Pathophysiologydo kwasa kansabe kolonbedǝ suroladǝn awowa kada mbeji, suroladǝn genetic mutations, inflammations, kuru awowa korbe.
Kǝndaram donyi tǝmatǝnyi madǝbe raksǝ kǝndaram donyi cellbe wuratǝ nzǝa banajin ma suwudin, kuru shilan cancer mbeji.
Awowa korbe jili kəmbu, kənəngatəbe-a, kuru kurunlaasobe raksə kasuwa kansa surobedəga suwudin.
Kәmbu buye tiyiye kime'a au dabbaye tiyin zәgana'a, misalro, kasuwa kansa surobegasoye'a kәlzәna.
Dalilla gade, jili nəm kǝrawu, tafa, kuru cidawa tiyibe ngǝlaro dio ba, sandi ye tajirwa'a dǝga fuzayin.
Sa kansa kansa surobedǝ wurazǝyinma, katapwa kada gojin, katap burobe-a doni suro kǝrǝnbe-an daata dǝga katapwa fuwuzanaa doni daji kansa dǝ nasha gadero tartǝyin ma.
Cellwa kansabe raksa tiyi'a karungube so'a naptəyin, kuru raksa lymphatic system men au bu men na'a cintu lan tartin, misallo tiyi liverbe au lungs so lan.
Kurunna donyi kansa kansa surobedǝ, shiro chemotherapy gultin, shiro radiation therapy gultin, adǝma dǝ lǝtǝram donyi kansa be ya.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
['Kәlanzәma ro cistәgә: liita']
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['Martәne netәram kәrәnbe do korowa dә ro jawawu suwudin dә, taganasmaro kalkal gәnyi sa lamba isәna dәn. Misallo, nәm nguwu am doni kwasa laa taganasbe shiro asuzana ma.']
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['(5) bayan nyima, kalima jirebero, ruwotәgәna, bayan suro watiyade dә kalkalzәna kuru nyiye hakku kәlanzәbe hakkuwa ləbtәma soye warmatәgәbe dәga nonәmma;']
['kuru (6) mukko dunowa au lantarkibe hakkiwa ləbtamabe au kam laaye wakil zəben cida sədənabe. ']
['Bayanna samibe samma kәltәgә nyiye mowonzә zәktә lamar buruwunәmye gәrәmin.']
['Lǝtǝram']
['Martәne ande ro watiya emailye lan koro/shawari laa manane.']
What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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