A patofixiologia do diabete a l"à da fâ co-o studio di proçesci fixiològichi desordinæ che pòrtan a-o sviluppo do diabete mellito.
O diabete mellito o l"é un gruppo de mouttie metaboliche caratterizzæ da di livelli erti de zucchero (glucòsio) into sangue che ne vëgnan da di difetti inta secreçion de l'insulina, inte l'açion de l'insulina ò inte tutte doe e mainee.
A patofixiologia do diabete a comprende l'interaçion complessa de fattoî genetichi, ambientali e do stile de vitta che contribuiscian a-o sviluppo da mouttia.
Into diabete de tipo 1, a patofisiologia a comprende unna distruçion autoimmune de çellole beta produte da l'insulina into pancreas, ch'a pòrta à unna mancansa de produçion de l'insulina.
Questo o l'à comme conseguensa l'incapaçitæ de regolare i livelli de glicemia into sangue, ch'o pòrta à l'iperglicemia (zuccaro into sangue erto).
Into diabete de tipo 2, a patofisiologia a l"é ciù complessa e a comprende tanto a rexistensa à l'insulina comme a segregaçion de l'insulina compromessa.
A rexistensa à l'insulina a l'intravëgne quande e çellole do còrpo no respòndan comme se deve à l'insulina, con portâ à unn'incapacitæ de deuviâ efficacemente o glucosio pe l'energia.
Sto fæto o fa produe into pancreas ciù insulina pe çercâ de superâ a rescistensa, ma co-o tempo o pancreas o no peu mantegnî o passo co-a domanda, con portâ à unna diminuçion da produçion de insulina.
O diabete gestaçionale, ch'o l'intravëgne inta gravidansa, o l'é caxonou da-i cangiamenti ormonali che pòrtan à unna rescistensa à l'insulina.
A placenta a produxe di ormoni che peuan bloccâ l'açion de l'insulina, con portâ à un aumento di livelli de glucosio into sangue.
A patofixiologia do diabete a comprende ascì o sviluppo de complicançe, comme danni a-i vasoin sanguigni, a-i nervi e a-i òrgani, che peuan portâ à di problemi de sanitæ gravi comme mouttie do cheu, ictus, mouttie di reni e perdia da vista.
A comprenscion da patofixiologia do diabete a l'é de cruçiâ importansa pe sviluppâ di trattamenti efficaxi e de strategie de prevençion pe sta mouttia cronica.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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