Pathophysiologie ya cancer ya poumon ezali kolobela mbongwana na misala ya physiologie mpe ba mécanisme oyo esalemaka na bokolisi mpe bokóli ya cancer ya poumon.
Cancer ya mimpululu ezali bokono ya makasi oyo ebimaka na bokóli mpe bokabwani ya ba cellules ya mabe na mimpululu.
Baselile yango ekoki kobimisa kanser mpe kopalangana na binama mosusu ya nzoto, mpe yango ekoki kobimisa bamaladi ndenge na ndenge.
Pathophysiologie ya cancer ya libale ezali na ba facteur ebele, na kati na yango ba mutation ya ba gène, ba facteur ya milieu mpe lolenge ya bomoi.
Mbongwana ya ba gène ekoki kosalema na ADN ya ba cellules ya poumon, oyo ekomema na bokóli ya ba cellules mpe bokabwani na yango.
Mbongwana yango ekoki kouta na makila to na maladi, mpe ekoki kouta na biloko oyo ebimisaka kanser, lokola milinga ya likaya, radon, amiante, mpe kobeba ya mopɛpɛ.
Cancer ya libale ekoki kokabolama na lolenge mibale ya minene: cancer ya libale ya cellule moke (SCLC) mpe cancer ya libale ya cellule moke te (NSCLC). NSCLC ekabolami lisusu na lolenge misato: adenocarcinome, squamous cell carcinoma, mpe large cell carcinoma.
Pathophysiologie ya lolenge oyo ya cancer ya libale ekoki kokesana, mpo bazali na ba mutation ya ba gene ekeseni mpe bakoyanola na ndenge ekeseni na nkisi.
Pathophysiologie ya cancer ya poumon ezali mpe na interaction kati na cellule ya cancer na tissus oyo ezali pembeni na yango, ata mpe système immunitaire.
Lisusu, microenvironnement ya tumeur ekoki kobakisa bokóli ya tumeur mpe metastase na kopesaka esika ya malamu mpo na ba cellules ya cancer.
Pathophysiologie ya cancer ya libale ezali likambo ya makasi mpe ya makasi, mpe bato ya mayele bazali kokoba kosala mpo na koyeba malamu ndenge ya kosala mpo na kobimisa nkisi ya malamu mpe kobongisa makambo ya bato ya maladi.
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Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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