La fisiopatologia del diabete la se riferiss al stüdi di prucess fisiologich disurdinà che pòrten al desvilup del diabete mellitus.
El diabete mellito l"è un grüp de malatì metaboliche caraterizad de livelli de zuccher (glucosio) in del sang elevad qe i derivan de difett ind la secrezion de l'insulina, ind l'azion de l'insulina o in tutt e duu.
La fisiopatologia del diabete la coinvolge la complessa interazion de fattur genetich, ambient e stil de vita qe contribuissen al desvilup de la malatia.
Ind el diabete de tipo 1, la fisiopatologia la coinvolge una distruziun autoimmune dei cellule beta che prodüsen insulina ind el pancreas, qe la porta a una deficenza ind la produziun de insulina.
Chesto el porta a un' incapacità de regulà i livelli de glucosi in del sangh, che porta a l' iperglicemia (aument de zuccher in del sangh).
Ind el diabete de tipo 2, la fisiopatologia l"è püsee cumplessa e la coinvolge sia la resistenza a l'insulina che la secrezion de l'insulina alterada.
La resistenza a l'insulina la succed quand i cellule del corp i respond minga ben a l'insulina, e l'impediss de duprà efficacement el glucosio per l'energia.
Chesto al fa che el pancreas al produca pü insulina per pruvà a superà la resistenza, ma cun el temp, el pancreas al pœl minga vess bon de tegnì el ritm de la dumanda, che la porta a una diminuziun de la produziun de insulina.
El diabete gestazional, qe al suced durant la gravidanza, l"è causad dei cambiament ormonai qe i porta a la resistenza a l'insulina.
La placenta la prodüss urmoni che pœden blocar l'azion de l'insulina, condüjend a un aument di livèll de glucosi in del sangh.
La fisiopatologia del diabete la coinvolge anca el desvilup de complicazion, cuma el dann ai vas sanguigni, ai nerv e ai orghen, qe pœden portar a seri problem de salute come malatie cardiache, ictus, malatie dei reni e perdita de vision.
Capir la fisiopatologia del diabete l"è crucial per sviluppà trattament e strategie de prevenzion efficaci per questa malattia cronica.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
['Disclaimer: medegh']
['Chestu sit web l"è furnid dumà per scopi educatif e informativ e l"è minga un cunsigli medich o un servizzi prufessiunal.']
["I informaziun furnid i g'han minga de vess drovad per diagnosticar o curar un prublema de salute o una malatia, e quei qe i cercan un cunsili medegh personal i g'han de consultar un medegh licenziad."]
['Per piasè, tegnì cont che la red neural che la genera rispost ai dumand l"è minga precisa quand che se tratta de numer, per esempi, el numer de gent che g\'ha una malattia precisa.']
["Cercà semper el cunsili del tò dutur o de un olter fornitur de sanità qualificaa in su una cundizion medica. Mai ignorà el cunsili medich prufessional o ritardà in de cercàll per via de un quaicoss che te gh'heet legiu in su'sto sit web. Se te penset de avègh una emergenza medica, ciama el 911 o va in del pronto soccors püssee vesin subit."]
['Disclaimer: copyright']
["El Digital Millennium Copyright Act del 1998, 17 U.S.C. § 512 (el DMCA) el dà recurs ai proprietari de copyright che creden che el material che 'l compariss in su Internet el infrange i sò dirit sota la legg di copyright di Stat Ünì. "]
["Se te credet de bona fede che un qualcun contegnü o material metüü a disposiziun in cunnessión cun el noster sit web o i noster servizzi infrang el tò copyright, te (o el tò agent) pudet mandà un avisi che dumanda che el contegnü o material vegn tolt, o che l'access a lü el vegn blocaa."]
['I avisi i g\'ha de vess mandaa per scrit per email (varda la sezion "Cuntatt" per l\'indirizz email).']
["La DMCA la dis che la tua notifica de presunta violazion del copyright la gh'ha de cuntà la seguent informazion: (1) descrizion de l'opera protetta dal copyright che l'è oget de la presunta violazion; (2) descrizion del presunt contegnud che 'l viola e informazion suficent per permetem de localizà el contegnud; (3) informazion de contatt per ti, cumprend el to indirizz, numer de telefon e indirizz email; (4) una declarazion de ti che te gh'heet bona fede che 'l contegnud de la manera reclamada l'è minga autorizzaa del proprietari del copyright, o del sò agent, o de la funzion de nissuna legg; "]
['(5) una declaraziun de ti, firmada sota pena de spergiur, che l\'informaziun in de la notificaziun l"è precisa e che te gh\'heet l\'autorità per fà valer i dirit de autor che se pretend de vess violaa;']
['e (6) una firma fisica o eletronica del proprietari del copyright o de una persona autorizada a agì per el proprietari del copyright.']
['Se no se inclüden tücc i infurmaziun sura indicà pœden causà un ritard in del processà la tua denuncia.']
['cuntatt']
['Per piasè mandem una mail cun qual-sa-vöör dumanda/suggeriment.']
What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
Disclaimer: medical
This web site is provided for educational and informational purposes only and does not constitute providing medical advice or professional services.
The information provided should not be used for diagnosing or treating a health problem or disease, and those seeking personal medical advice should consult with a licensed physician.
Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
Always seek the advice of your doctor or other qualified health provider regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website. If you think you may have a medical emergency, call 911 or go to the nearest emergency room immediately. No physician-patient relationship is created by this web site or its use. Neither BioMedLib nor its employees, nor any contributor to this web site, makes any representations, express or implied, with respect to the information provided herein or to its use.
Disclaimer: copyright
The Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the “DMCA”) provides recourse for copyright owners who believe that material appearing on the Internet infringes their rights under U.S. copyright law. If you believe in good faith that any content or material made available in connection with our website or services infringes your copyright, you (or your agent) may send us a notice requesting that the content or material be removed, or access to it blocked. Notices must be sent in writing by email (see 'Contact' section for email address) . The DMCA requires that your notice of alleged copyright infringement include the following information: (1) description of the copyrighted work that is the subject of claimed infringement; (2) description of the alleged infringing content and information sufficient to permit us to locate the content; (3) contact information for you, including your address, telephone number and email address; (4) a statement by you that you have a good faith belief that the content in the manner complained of is not authorized by the copyright owner, or its agent, or by the operation of any law; (5) a statement by you, signed under penalty of perjury, that the information in the notification is accurate and that you have the authority to enforce the copyrights that are claimed to be infringed; and (6) a physical or electronic signature of the copyright owner or a person authorized to act on the copyright owner’s behalf. Failure to include all of the above information may result in the delay of the processing of your complaint.
['Circa']
['BioMedLib dupera computer automatizzads (algoritmi de machine-learning) per generar coppie de dumanda e risposta.']
['Cominciom con 35 milion de publicazion biomediche de PubMed/Medline.']
