Pathophysiology mar kansa mar colon wuoyo kuom gik matimore kod gik matimore ma kelo dongruok kod dongruok mar kansa mar colon.
Tuwo mar kansa mar colon, ma bende iluongo ni colorectal cancer, en kit tuwo mar kansa ma chakore e colon kata rectum, ma gin kuonde mag del maduong'.
Kinde mang'eny, tuwono chakore gi ng'injo miluongo ni polyp, ma bang' kinde nyalo bedo kansa.
Tuwo mar kansa mar colon oriwo gik mang'eny, moriwo nyaka lokruok ma timore e dend ng'ato, tuwo mar ng'ol, kod gik matimore e alwora.
Lokruok mag anyuola nyalo timore e DNA mar ng'injo mag colon, kendo mano nyalo miyo ng'injo omed dongo kendo pogore e yo ma ok nyal geng'.
Lokruok ma kamago nyalo bedo mar anyuola kata mar ng'ato owuon, kendo ginyalo hinyo gik moko mopogore opogore motudore gi dongo mar ng'injo, pogo ng'injo, koda loso gik moko.
Tuwo mar colon, ma nyalo bedo nikech tuoche kaka inflammatory bowel disease, bende nyalo miyo ng'ato obed gi kansa mar colon.
Tuwo ma ok rum nyalo kelo gik moko ma miyo ng'injo dongo kendo pogore, kendo mano nyalo miyo ng'ato obed gi tuwo mar kansa.
Gik matimore e alwora, kaka chiemo ma ng'ato chamo, kit ngima ma ng'ato odakie, koda tiyo gi yedhe moko, bende nyalo miyo ng'ato obed gi kansa mar colon.
Kuom ranyisi, chiemo mang'eny moting'o ring'o makwar kod ring'o molos maber, osemiyo ng'ato bedo gi tuwo mar kansa mar colon.
Gik mamoko kaka bedo gi del mapek, madho ndawa, kod bedo ma ok ti gi del mang'eny, bende nyalo miyo ng'ato obed gi tuoche mang'eny.
Bang' ka kansa mar colon osechako bedoe, onyalo dhi nyime e okang' mopogore opogore, chakre e okang' mokwongo mar kansa mar colon nyaka e okang' machielo ma kansa oselandore e fuonde mamoko mag del.
Ng'injo mag kansa nyalo monjo fuonde koda fuonde mag del man machiegni, kendo ginyalo landore kokalo kuom lymphatic system kata remo nyaka kuonde mabor, kaka ich kata fuonde.
Kinde mang'eny, thieth mar kansa mar colon oriwo yeng'o, chemotherapy, kod radiation therapy, kaluwore gi kama tuwo mar kansa yudoree.
Fwenyo tuo mar kansa chon kod thiedhe en gima duong' ahinya e miyo tuwono obed maber, nimar kinde mang'eny inyalo thiedhe kapo ni oyud tuwono e okang' motelo.
Nonruok kinde ka kinde, kaka timo colonoscopy, nyalo konyo e fwenyo kansa mar colon e okang' mokwongo, e kinde ma inyalo thiedhe maber.
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Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
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What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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