Pathophysiology mar tuwo mar sukari ochung' ne puonjruok mar yore mag del ma ok ochanore ma kelo tuwo mar sukari.
Tuwo mar sukari (diabetes mellitus) en grup mar tuoche mag metabolic ma nyiso ni ng'ato nigi sukari mang'eny e remo (glucose) nikech tuwo mar insulin, kata nikech tuwo mar insulin, kata nikech tuoche ariyogo.
Tuwo mar sukari oriwo gik mang'eny motudore gi anyuola, chal mar piny, koda kit ngima ma miyo ng'ato bedo gi tuwo mar sukari.
E tuwo mar sukari mar type 1, tuwo mar pathophysiology kelo kethruok mar ng'injo mag beta ma loso insulin e pancreas, kendo mano miyo ng'ato ok nyal loso insulin moromo.
Mano miyo ng'ato ok nyal chiko teko mar glucose e remo, to mano miyo ng'ato bedo gi hyperglycemia (mero mar sukari e remo).
E tuwo mar sukari miluongo ni type 2, chal mar del mar ng'ato ok yot ng'eyo, kendo en gi tuwo mar insulin resistance kod tuwo mar insulin secretion.
Insulin geng'o insulin sama ng'injo mag del ok winj insulin e yo makare, to mano miyo ok ginyal tiyo gi glucose e yo maber mondo giyud teko.
Mano miyo pancreas loso insulin mang'eny mondo otem loyo temno, kata kamano, bang' kinde, pancreas nyalo bedo ni ok nyal chopo dwaro mar insulin, to mano nyalo miyo insulin kik bed mang'eny.
Tuwo mar sukari mar nyuol, ma timore e kinde ma ng'ato en gi ich, en gima timore nikech lokruok ma timore e hormone mamiyo ng'ato ok nyal geng'o tiyo gi insulin.
Placenta golo hormone ma nyalo geng'o insulin kik ti, to mano miyo remo bedo gi glucose mang'eny.
Tuwo mar sukari bende nyalo kelo chandruoge moko, kaka hinyruok mar choke mag remo, obwongo, kod fuonde mamoko mag del, ma nyalo kelo chandruoge madongo kaka tuoche mag chuny, tuwo mar strok, tuoche mag nyiroke, koda lalo wang'.
Ng'eyo kaka tuwo mar sukari timore en gima duong' ahinya e loso yore mag thiedho tuwo mar sukari kendo geng'o tuwono kik timre.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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