Pathophysiology mar kansa mar obo wuoyo kuom lokruoge matimore e yore mapile mag del kod yore matimore e dongruok kod dongruok mar kansa mar obo.
Tuwo mar kansa mar obwongo en tuwo ma kelo tuoche mang'eny nikech ng'eny mar ng'injo mag obwongo ma ok nyal geng' kendo ma pogore.
Ng'injo ma kamago nyalo bedo gi tuwo mar ndulme kendo landore e fuonde mamoko mag del, to mano nyalo miyo ng'ato obed gi tuoche mopogore opogore.
Tuwo mar kansa mar obo otenore kuom gik mang'eny, moriwo nyaka lokruok ma timore e dend ng'ato, gik matimore e alwora, koda kit ngima ma ng'ato luwo.
Lokruok mag anyuola nyalo timore e DNA mar ng'injo mag ofuko, kendo mano nyalo miyo ng'injo omed dongo kendo pogore e yo ma ok nyal geng'.
Lokruok ma kamago nyalo bedo mar anyuola kata mar ng'ato owuon, kendo ginyalo bedoe nikech tiyo gi gik ma kelo kansa, kaka iro mar ndawa, radon, asbestos, kod muya mokethore.
Tuwo mar kansa mar obwongo inyalo pog e yore ariyo madongo: small cell lung cancer (SCLC) kod non-small cell lung cancer (NSCLC). NSCLC bende ipogo e yore adek: adenocarcinoma, squamous cell carcinoma, kod large cell carcinoma.
Kit kansa mag obwongo ma kamago nyalo bedo mopogore opogore nikech gin gi gik mopogore opogore e dendgi, kendo ginyalo dwoko thieth e yo mopogore.
Tuwo mar kansa mar obo bende oriwo tudruok mantie e kind ng'injo mag kansa kod ng'injo molworogi, moriwo koda ng'injo mag geng'o tuoche.
Ng'injo mag kansa nyalo ringo geng'o teko mar geng'o tuoche, kendo mano miyo ng'injo mag kansa dongo kendo landore ma onge ng'ama geng'ogi.
E wi mano, microenvironment mar tumor nyalo miyo tumor omed dongo kendo kelo metastasis kuom chiwo alwora maber ne ng'injo mag kansa.
Tuwo mar kansa mar obo en gima tek ng'eyo, kendo josayans tiyo matek mondo ging'e kaka tuwono timore, mondo giyud yore mag thiedho tuwono e yo maber moloyo, kendo mondo gikony jatuwo.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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Weche mochiw ok onego oti kodgi e fwenyo kata thiedho tuwo moro, kendo jogo madwaro ng'eyo kaka ginyalo thiedho tuwo moro, onego owuo gi laktar molony.
Ng'e ni neural net ma chiwo dwoko mag penjo, ok en makare ahinya sama iwuoyo kuom kwan mag ji, kaka kwan mar joma nigi tuwo moro.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
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