Patofisiologi diabetes marujuak pado kajian proses fisiologis nan indak teratur nan manyababkan tajadinyo diabetes mellitus.
Diabetes mellitus adolah sakalompok panyakik metabolik nan ditandoi jo tingkek gulo darah (glukosa) nan tinggi nan disababkan dek cacat sekresi insulin, aksi insulin, atau kaduonyo.
Patofisiologi diabetes maliputi interaksi kompleks faktor genetik, lingkungan, jo gaya hiduik nan manyababkan panyakik ko.
Pado diabetes tipe 1, patofisiologi maliputi karusakan otoimun dari sel beta nan mahasiakan insulin di pankreas, nan manyababkan kakurangan produksi insulin.
Hal iko manyababkan indak dapek maatur kadar glukosa darah, nan manyababkan hiperglikemia (tinggi gulo darah).
Pado diabetes tipe 2, patofisiologi labiah kompleks jo maliputi resistensi insulin jo gangguan sekresi insulin.
Hal iko manyababkan pankreas mamproduksi labiah banyak insulin untuak mancubo mangalahkan resistensi, tapi sairiang wakatu, pankreas mungkin indak dapek maisi permintaan, nan manyababkan panurunan produksi insulin.
Diabetes gestasional, nan tajadi salamo manganduang, disababkan dek parubahan hormon nan manyababkan resistensi insulin.
Plasenta mahasiakan hormon nan dapek mamblokir aksi insulin, nan manyabaukan tingkek glukosa darah maningkek.
Patofisiologi diabetes juo maliputi pakambangan komplikasi, sarupo karusakan pado pambuluah darah, saraf, jo organ, nan dapek manyababkan masalah kasihatan serius sarupo panyakik jantung, stroke, panyakik ginjal, jo ilang pancaliakan.
Mamahami patofisiologi diabetes pantiang untuak mangambangan pangobatan jo strategi pancegahan nan efektif untuak panyakik kronis iko.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
['Disclaimer: medis']
['Situs web iko hanyo digunokan untuak tujuan pandidikan jo informasi sajo dan indak mambari nasehat medis atau layanan profesional.']
['Informasi nan disadioan indak buliah digunoan untuak mandiagnosis atau maubek masalah kasihatan atau panyakik, dan urang nan mancari nasehat medis pribadi harus bakonsultasi jo dotor nan balisensi.']
['Tolong diingek jaringan saraf nan manghasilkan jawaban untuak pertanyaan, khususnyo indak akurat katiko datang ka isi numerik. contohnyo, jumlah urang nan didiagnosis jo panyakik tatantu.']
['Salalu mintak nasehat dotor atau panyadia kasihatan lain nan kualifikasi manganai kondisi medis. Jan pernah maabaikan nasehat medis profesional atau talambek dalam mamintaknyo dek karano sasuatu nan awak baco di situs web iko. Jiko awak sangko mungkin ado kaadaan darurat medis, hubungi 911 atau pai ka ruang gawat darurat paliang dakek lansuang. Indak ado hubungan dotor-pasien nan diciptaan dek situs web iko atau panggunoannyo. BioMedLib maupun karyawan, maupun kontributor situs web iko, indak ado malakukan representasi, jaleh atau tasuruak, manganai informasi nan disadioan di siko atau panggunoannyo.']
['Panyalasaian: hak cipta']
['Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (DMCA) manyadiokan jalan bagi pamilik hak cipta nan picayo bahwasanyo bahan nan muncul di Internet malangga haknyo manuruik hukum hak cipta AS. ']
['Jiko Sanak yakin jo yakin baso isi atau bahan nan tasadio dalam pautan ka situs web atau layanan kami malangga hak cipta Sanak, Sanak (atau agen Sanak) dapek mangirim kami pasan mamintak untuak manghapuih isi atau bahan tasabuik, atau mamblokir akses ka situs tasabuik. ']
['Kabar harus dikirim sacaro tulisan malalui surel (caliak bagian "Kontak" untuak alamat surel).']
['DMCA mamaraluan supayo pasan pasan palanggaran hak cipta nan didakwa tamasuak informasi barikuik: (1) deskripsi karya hak cipta nan manjadi subyek palanggaran nan didakwa; (2) deskripsi isi nan didakwa malanggar jo informasi nan cukuik untuak mamungkinkan kami untuak manamukan isi; (3) informasi kontak untuak Sanak, tamasuak alamat, nomor telepon jo alamat surel; (4) pernyataan Sanak baso Sanak punyo kayakinan baiak baso isi nan dikaduan indak diizinkan dek nan punyo hak cipta, atau agennyo, atau dek operasi hukum apo pun; ']
['(5) suatu pernyataan nan ditandotangani dek Sanak, jo hukuman sumpah palsu, baso informasi dalam notifikasi tu akurat jo Sanak punyo wewenang untuak manjalankan hak cipta nan dikatokan alah di langgar; ']
['dan (6) ciek tando tangan fisik atau elektronik dari pamilik hak cipta atau sasaurang nan punyo hak untuak batindak ateh namo pamilik hak cipta. ']
['Kok indak tamasuak sado informasi di ateh dapek manyababkan panundaan pangolahan aduan Sanak.']
['Kontak']
['Tolong kirim surel untuak pertanyaan/saran.']
What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
Disclaimer: medical
This web site is provided for educational and informational purposes only and does not constitute providing medical advice or professional services.
The information provided should not be used for diagnosing or treating a health problem or disease, and those seeking personal medical advice should consult with a licensed physician.
Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
Always seek the advice of your doctor or other qualified health provider regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website. If you think you may have a medical emergency, call 911 or go to the nearest emergency room immediately. No physician-patient relationship is created by this web site or its use. Neither BioMedLib nor its employees, nor any contributor to this web site, makes any representations, express or implied, with respect to the information provided herein or to its use.
Disclaimer: copyright
The Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the “DMCA”) provides recourse for copyright owners who believe that material appearing on the Internet infringes their rights under U.S. copyright law. If you believe in good faith that any content or material made available in connection with our website or services infringes your copyright, you (or your agent) may send us a notice requesting that the content or material be removed, or access to it blocked. Notices must be sent in writing by email (see 'Contact' section for email address) . The DMCA requires that your notice of alleged copyright infringement include the following information: (1) description of the copyrighted work that is the subject of claimed infringement; (2) description of the alleged infringing content and information sufficient to permit us to locate the content; (3) contact information for you, including your address, telephone number and email address; (4) a statement by you that you have a good faith belief that the content in the manner complained of is not authorized by the copyright owner, or its agent, or by the operation of any law; (5) a statement by you, signed under penalty of perjury, that the information in the notification is accurate and that you have the authority to enforce the copyrights that are claimed to be infringed; and (6) a physical or electronic signature of the copyright owner or a person authorized to act on the copyright owner’s behalf. Failure to include all of the above information may result in the delay of the processing of your complaint.
['Sakitarnyo']
['BioMedLib manggunokan komputer otomatis (algoritma machine-learning) untuak mambuek pasangan pertanyaan-jo-jawaban.']
['Kami mamulai jo 35 juta publikasi biomedis dari PubMed/Medline. juo, laman web dari RefinedWeb.']
