What is pathophysiology of Lung cancer?

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X'inhi l- patofiżjoloġija tal- kanċer tal- pulmun?

Il- patofiżjoloġija tal- kanċer tal- pulmun tirreferi għall- bidliet fil- proċessi u l- mekkaniżmi fiżjoloġiċi normali li jseħħu fl- iżvilupp u l- progressjoni tal- kanċer tal- pulmun.

Il- kanċer tal- pulmun huwa marda kumplessa li tirriżulta mit- tkabbir u d- diviżjoni mhux ikkontrollati ta ' ċelloli anormali fil- pulmun.

Dawn iċ- ċelloli jistgħu jiffurmaw tumuri u jinfirxu f' partijiet oħra tal- ġisem, u dan iwassal għal diversi sintomi u kumplikazzjonijiet.

Il- patofiżjoloġija tal- kanċer tal- pulmun tinvolvi diversi fatturi, inklużi mutazzjonijiet ġenetiċi, fatturi ambjentali, u għażliet fl- istil taʼ ħajja.

Jistgħu jseħħu mutazzjonijiet ġenetiċi fid- DNA taċ- ċelloli tal- pulmun, li jwasslu għal tkabbir u diviżjoni taċ- ċelloli mhux ikkontrollati.

Dawn il- mutazzjonijiet jistgħu jiġu ereditati jew akkwistati, u jistgħu jiġu kkawżati minn esponiment għal karċinoġeni, bħal duħħan tat- tabakk, radon, asbestos, u tniġġis tal- arja.

Il-kanċer tal-pulmun jista 'jiġi kklassifikat f'żewġ tipi ewlenin: kanċer tal-pulmun b'ċelloli żgħar (SCLC) u kanċer tal-pulmun b'ċelloli mhux żgħar (NSCLC). NSCLC huwa maqsum aktar fi tliet sottotipi: adenokarcinoma, karcinoma ta' ċelloli squamous, u karcinoma ta 'ċelloli kbar.

Il- patofiżjoloġija ta ' dawn it- tipi ta ' kanċer tal- pulmun tista ' tkun differenti, peress li għandhom mutazzjonijiet ġenetiċi differenti u jirrispondu b' mod differenti għat- trattament.

Il- patofiżjoloġija tal- kanċer tal- pulmun tinvolvi wkoll l- interazzjoni bejn iċ- ċelloli tal- kanċer u t- tessut tal- madwar, inkluż is- sistema immunitarja.

Iċ- ċelloli tal- kanċer jistgħu jevitaw is- sistema immunitarja, u b'hekk ikunu jistgħu jikbru u jinfirxu mingħajr kontroll.

Barra minn hekk, il- mikroambjent tat- tumur jista ' jippromwovi t- tkabbir tat- tumur u l- metastażi billi jipprovdi ambjent ta ' appoġġ għaċ- ċelloli tal- kanċer.

Il- patofiżjoloġija tal- kanċer tal- pulmun hija proċess kumpless u dinamiku, u r- riċerkaturi qed jaħdmu kontinwament biex jifhmu aħjar il- mekkaniżmi sottostanti biex jiżviluppaw trattamenti aktar effettivi u jtejbu r- riżultati tal- pazjent.

Referenzi

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.

Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.

Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.

Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .

Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .

Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.

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Jekk temmen b'bona fidi li kwalunkwe kontenut jew materjal magħmul disponibbli b'rabta mal-websajt jew is-servizzi tagħna jikser id-drittijiet tal-awtur tiegħek, inti (jew l-aġent tiegħek) tista' tibgħatilna avviż li jitlob li l-kontenut jew il-materjal jitneħħa, jew li l-aċċess għalih jiġi mblukkat.

L-avviżi għandhom jintbagħtu bil-miktub bil-posta elettronika (ara t-taqsima "Kuntatt" għall-indirizz tal-posta elettronika).

Id-DMCA teħtieġ li n-notifika tiegħek ta' ksur allegat tad-drittijiet tal-awtur tinkludi l-informazzjoni li ġejja: (1) deskrizzjoni tax-xogħol protett bid-drittijiet tal-awtur li huwa s-suġġett ta' ksur allegat; (2) deskrizzjoni tal-kontenut allegatament li jikser id-drittijiet tal-awtur u informazzjoni suffiċjenti biex inkunu nistgħu nsibu l-kontenut; (3) informazzjoni ta' kuntatt għalik, inkluż l-indirizz tiegħek, in-numru tat-telefon u l-indirizz tal-email; (4) dikjarazzjoni minnek li għandek twemmin ta' fidi tajba li l-kontenut bil-mod li dwaru l-ilment ġie ppreżentat mhuwiex awtorizzat mid-detentur tad-drittijiet tal-awtur, jew mill-aġent tiegħu, jew mill-operazzjoni ta' kwalunkwe liġi;

(5) stqarrija minnek, iffirmata taħt il-piena ta' perjuri, li l-informazzjoni fin-notifika hija preċiża u li għandek l-awtorità li tinforza d-drittijiet tal-awtur li huma ddikjarati li ġew miksura;

u (6) firma fiżika jew elettronika tas-sid tad-drittijiet tal-awtur jew ta' persuna awtorizzata li taġixxi f'isem is-sid tad-drittijiet tal-awtur.

In-nuqqas li tinkludi l-informazzjoni kollha ta' hawn fuq jista' jwassal għal dewmien fit-trattament tal-ilment tiegħek.

Kuntatt

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What is pathophysiology of lung cancer?

The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.

Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.

These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.

The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.

Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.

These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.

Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.

The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.

The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.

Cancer cells can evade the immune system, allowing them to grow and spread unchecked.

Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.

The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.

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