What is pathophysiology of Heart attack?

Ɛŋu mi̱ la pathophysiology duŋ lök lɔaac?

Kä pathophysiology duŋ lök lɔaac, mi̱ ŋa̱ckɛ bä cie mi̱o̱kardial inpa̱rkciɔn, cuɛ te rɛy kä ɣöö ci̱ riɛm wä rɛy puɔ̱nydɛ, min cu nööŋ ni̱ lia̱a̱ thëlli̱ lo̱c.

Nɛmɛ laa tuɔɔk kɛ ɣöö laa tuɔɔm riɛm mi̱ ci̱ rɔ̱ yiath piny laa pën duɔ̱ɔ̱p in laa jäl kɛ riɛm, kä laa tuɔɔm kɛ kui̱i̱ atheri̱thklɛrɔthith, ɛ ta̱a̱ in laa rɔ̱ laa täthkɛ kɛ mi̱ ci̱ rɔ̱ laa luäŋ kɛ kuëŋ rɛy duɔ̱ɔ̱p in laa jäl kɛ riɛm.

Kä pla̱k ɛ mi̱ ca la̱t kɛ kɔlɛthtɛrɔl, mi̱ te rɛy pua̱a̱ny, mi̱ la̱tkɛ ɛ thëlli̱, kalci̱öm, kɛnɛ pi̱bi̱ri̱n.

Mi̱ ci̱ pla̱k rɔ da̱a̱k, derɛ riɛm mi̱ ci̱ rɔ luäŋ luäŋ kɛ rɔ̱ɔ̱diɛn jakä bi̱ rɔ̱ɔ̱diɛn cu rɔ̱ɔ̱diɛn luäŋ kɛ rɔ̱ɔ̱diɛn, min bi̱ arteri̱ cu gäk kä bi̱ riɛm mi̱ ci̱ rɔ̱ɔ̱diɛn luäŋ kɛ yiëë mi̱ di̱i̱t kä ɣɔkdhi̱ji̱n gaŋ kɛ ɣöö bɛ cop kä lo̱o̱ny lɔaac.

Ɛn thiɛl ɔksi̱ji̱n ɛmɛ jakɛ thëlli̱ tin tä rɛy lo̱o̱ny lɔaac kä li̱w, kä no̱o̱ŋɛ kɛ kä ɣöö bi̱ lɔcdɛ cu te kɛ riɛk.

Kä pek in ci̱ duäc thi̱n cuɛ tekä pek in ci̱ duäc thi̱n kä cu duäcdɛ te thi̱n kä cu te kɛ gua̱a̱th mi̱ bä kam kä mëë ci̱ duäc kɛnɛ min ca luäk.

Nyuthni̱ tin la̱tkɛ kɛ juey lɔaac de̱e̱ te rɛy lo̱cni̱ ti̱ bɛc kiɛ bɛc, yiëë mi̱ ci̱ rɔ bɛc, jiɛth, wi̱c mi̱ ci̱ rɔ yi̱c, kɛnɛ bɛc pua̱a̱ny, ŋua̱l, lɛ̈ɛ̈ŋ, kiɛ jɔk.

Kä ta̱a̱ in laa jiek kɛ jɛ kɛ juey lɔaac laa lotɛ ni̱ ɣöö bi̱ riɛm rɔ nyɔk kɛ loc rɛy lo̱o̱nyä lɔaac kɛ pɛ̈th, kɛ wal kiɛ kɛ ca̱a̱p ti̱ ciee kɛ angioplasti̱ kɛnɛ stenting kiɛ coronary artery bypass surgery.

Jɛn ɛ mi̱ di̱i̱t ɛn ɣöö ba ŋa̱c ɛn ɣöö ɛn pato̱pi̱thi̱ölöji̱ duŋ lök lɔaac ɛ mi̱ räth kä matdɛ ŋɔak ti̱ ŋuan, amäni̱ ji̱i̱n, ta̱a̱ teekä, kɛnɛ tin te rɛy gua̱th in cieŋ raan thi̱n.

Kɛn tin dee raan jakä bɛc kɛ juey lɔcdɛ ɛ mi̱ cie riɛm mi̱ di̱i̱t, cholesterol mi̱ di̱i̱t, täth kɛ wal, juey thɛktɔrä, di̱t pua̱a̱ny, /thiɛl lät pua̱a̱ny, kɛnɛ tëë ci̱ tuɔɔk ni̱ wal rɛy gɔaalä mi̱ tekɛ juey lɔc.

Mi̱ ci̱ riɛk ɛmɛ cop kä ji̱n derɛ luäk kɛ ɣöö bi̱ cu ben piny kä ɣöö bi̱ cu ben kɛ juey lɔaac.

['Kuënɛ']

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.

Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .

Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.

LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.

Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.

Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.

['Lät kɛ kui̱i̱ jua̱thni̱']

['Ɛn wɛbthaay ɛmɛ ca la̱th lät kɛ kui̱ ŋi̱i̱cä kɛnɛ läri kä /cɛ lot ni ɣöö ba ji̱ moc kɛ luäk ki̱mä kiɛ lät ti̱ gööl.']

['Lät kɛ läri tin ca ŋun /ca kɛ bi̱ lät kɛ ɣöö ba jua̱th jek kiɛ ba kɛ ciɛŋ, kä nɛy tin görkɛ luäk ki̱mädiɛn kärɔ̱ ba kɛ thiec kä ki̱m mi̱ tekɛ luäk ki̱mä.']

['Guic ɛ gɔaa ɛn ɣöö ɛn neural net min jak luɔc kä thie̱cni̱, /cɛ thuɔ̱k ɛlɔ̱ŋ mi̱ ci̱ ben kä nämbäri̱ tin te thi̱n. cetkɛ pek nath tin ca jek kɛ juey mi̱ rɛlrɔ.']

['Ni ciaŋ go̱ri ruac kä ki̱mdu kiɛ ram in kɔ̱ŋ mi ŋäc luäk pua̱a̱ny kɛ kui̱ jua̱thdu. /Cu ruac ki̱mdu car kiɛ jääny kɛ go̱ri kɛ kui̱ kä mi ci kuɛn kä wɛbthaay ɛmɛ. Mi caari jɛ ɛn ɣöö deri tekɛ juey mi go̱o̱ri luäk, cɔl 911 kiɛ wër guäth in thia̱k kɛ ji kɛ pɛ̈th. /Thilɛ maar kam ki̱m kɛnɛ juey mi bi̱ tuɔɔk kɛ kui̱ kä wɛbthaay ɛmɛ kiɛ la̱tdɛ. /Ci̱ BioMedLib kiɛ la̱a̱tkɛ, kiɛ ram in gɔ̱a̱r kɛ kui̱ kä wɛbthaay ɛmɛ, bi̱ ruac lat, kiɛ bi̱ ruac lat, kɛ kui̱ läri tin ca ka̱m raar rɛydɛ kiɛ la̱tdɛ.']

['Lät kɛ: ŋuɔ̱t']

['Ɛn Digital Millennium Copyright Act 1998, 17 U.S.C. § 512 (ɛ DMCA) ɛ ŋuɔ̱t mi̱ ŋun ji̱ cuŋni̱ tin ŋääth kɛn ɣöö ci̱ ŋɔaani̱ tin te kä intɛrnɛt ŋuɔ̱tkiɛn to̱l kɛ kui̱ ŋuɔ̱tni̱ cuŋni̱ tin te kä U.S.']

['Mi ca ji̱ ŋäth kɛ thuɔ̱k ɛn ɣöö tëëkɛ mi̱ ca gɔ̱r kiɛ mi̱ ca ka̱m ji̱ rɛy wɛbthaayäda kiɛ lät tin kɔ̱ŋ tin ci̱ ŋuɔ̱t tin ca gɔ̱r ya̱r, ji̱n (kiɛ ram in lät kɛ kui̱du) deri̱ kɔ jäk kä warɛgak mi̱ bi̱ ji̱ thiec ɛn ɣöö ba min ca gɔ̱r kiɛ min ca gɔ̱r woc, kiɛ ba duɔ̱ɔ̱r la̱t kɛ ɣöö bi̱ ji̱ cop thi̱n.']

['Kɛn läri̱ ba kɛ jäk kɛ wargak ɛ la i̱thtäm (guic ni̱ gua̱th in ci̱ i̱thtäm in ca gɔ̱r "Kɔntak" kɛ kui̱ i̱thtäm in ci̱ jäk).']

['DMCA go̱o̱rɛ ɣöö bi̱ ji̱n warɛgakdu lat kɛ kui̱ kä tin ca lar i̱ ci̱ ŋuɔ̱t gɛr kɛ kui̱i̱ ŋuɔ̱tni̱ gɔ̱rä mat thi̱n kɛ läär ti̱ti̱: (1) latdɛ kɛ kui̱i̱ la̱t in ca gɛr kɛ kui̱i̱ ŋuɔ̱tni̱ gɔ̱rä min ca lar i̱ ca gɛr; (2) latdɛ kɛ kui̱i̱ kä tin ca lar i̱ ci̱ ŋuɔ̱t gɛr kɛnɛ läär ti̱ ro̱ŋ kɛ ɣöö bi̱ kɔn kɛ jek; (3) läri̱ kɛ kui̱i̱ kä min dëë ji̱ luäk kɛ jek, amäni̱ ci̱ötdu, nämbärɛ kä tin ci̱ ji̱ luäk kɛ jek, kɛnɛ emaildu; (4) latdu kɛ ɣöö ci̱ ji̱n ɛ ŋa̱c ɛn ɣöö min ca gɔ̱r kɛ kui̱i̱ kä tin ca lar /ci̱kɛ bi̱ lät kɛ luäkdɛ ɛ gua̱n ŋuɔ̱tni̱ gɔ̱rä, kiɛ ɛ la̱tdɛ, kiɛ kɛ luäk ŋuɔ̱tni̱; ']

['(5) mi ci ji gɔr piny kɛ kuic kä ɣöö bi ji ruac kɛ thuɔ̱k, ɛn ɣöö läri tin ca gɔr ɛ thuɔ̱k kä te ji kɛ lua̱ŋ kɛ ɣöö bi yiöw tin ca gɔr piny tin ca lar ɛ ji̱n ka̱m raar;']

['kɛnɛ (6) mi̱ ca gɔ̱r piny ɛ gua̱n ŋuɔ̱tni̱ kiɛ ram mi̱ tekɛ lua̱ŋ kɛ lät kɛ kui̱dɛ.']

['Mi /kenɛ läri tin ca lat nhial diaal mat thi̱n dɔ̱ŋ derɛ ku lɛ wɔ̱ jɔ̱ɔ̱r kɛ lätni̱ kɛ kɛ.']

['Röm kɛ jɛ']

['Thiecɛ kɔ kɛ email kɛ thiecni kiɛ cär.']

What is pathophysiology of heart attack?

The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.

This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.

The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.

When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.

This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.

The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.

Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.

Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.

It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.

Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.

Managing these risk factors can help reduce the likelihood of experiencing a heart attack.

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