Kodi pathophysiology ya khansa ya m'matumbo ndi yotani?
Pathophysiology ya khansa ya m'matumbo imatanthauza njira ndi njira zomwe zimayambitsa chitukuko ndi kupita patsogolo kwa khansa ya m'matumbo.
Khansa ya m'matumbo aakulu, yomwe imadziwikanso kuti khansa ya m'matumbo aakulu, ndi mtundu wa khansa imene imayamba m'matumbo aakulu kapena m'matumbo aakulu, omwe ndi mbali za m'matumbo aakulu.
Kaŵirikaŵiri imayamba monga kukula kotchedwa polyp, kumene kungasanduke kansa m'kupita kwa nthaŵi.
Matenda a khansa ya m'matumbo amaphatikizapo zinthu zingapo, kuphatikizapo kusintha kwa majini, kutupa, ndi zinthu za m'chilengedwe.
Kusintha kwa majini kungachitike mu DNA ya maselo a m'matumbo aakulu, kuchititsa kukula kwa maselo ndi kugaŵikana kosalamulirika.
Kusintha kwa maselo kumeneku kungakhale kwachibadwa kapena kwachilengedwe, ndipo kungakhudze majini osiyanasiyana okhudzidwa ndi kukula kwa maselo, kugaŵikana, ndi kukonzanso.
Matenda a m'matumbo aakulu, amene angayambitsidwe ndi matenda monga matenda a m'matumbo aakulu, angathandizenso kuti munthu akhale ndi khansa ya m'matumbo aakulu.
Khansa ya m'matumbo ikangoyamba kumene, imatha kupita m'magawo angapo, kuyambira ku khansa yoyambirira imene imangokhala m'matumbo mpaka ku khansa ya m'matumbo imene yafalikira m'madera ena a thupi.
Maselo a khansa angaloŵe m'matumbo ndi m'ziwalo zapafupi, ndipo angafalikire kupyolera m'dongosolo la lymphatic kapena m'magazi ku malo akutali, monga chiwindi kapena mapapo.
Kaŵirikaŵiri chithandizo cha khansa ya m'matumbo chachikulu chimaphatikizapo opaleshoni, chemotherapy, ndi radiation therapy, malinga ndi siteji ndi malo a khansa.
Kuzindikira ndi kuchiza matenda msanga n'kofunika kwambiri kuti matendawa ayende bwino, chifukwa chakuti khansa ya m'matumbo imatha kuchiritsidwa ikangopezeka koyambirira.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
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Chonde titumizireni imelo ndi funso / lingaliro lililonse.
What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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