Say pathophysiology na prostate cancer et ontutukoy ed saray manunan proseso ya sengegan na iyaaligwas na sakit.
Say prostate cancer et malignant tumor ya ompapaway ed saray selula na prostate gland, ya melag tan singa walnut ya organ ya walad leksab na bladder na laki.
Say prostate gland so mamapawala na seminal fluid, a mamapabuna tan mangitatarok na saray sperm.
Agni sigpot a natatalosan no anton talagay panlalapuan na prostate cancer, balet wala ray pigaran rason no akin ya ondarakel so posibilidad ya nawalaan na satan.
Kabiangan ed saratan so edad, pamilya, lahi, tan pigaran mutation ed genes.
Mas mabetbet so prostate cancer ed saray mas matatken a lalaki, a maslak ed saray kaso et diad saray mas matatken nen 65.
Niarum ni, saray lalakin walay pamilya ran walay prostate cancer et mas baleg so posibilidad ya nawalaan, ontan met ed saray lalakin African American tan Caribbean.
Say pathophysiology na prostate cancer et nalalanor so agnakontrol ya ibaleg tan panlalaok na selula ed prostate gland.
Nayarin nagawa iya lapud genetic mutations ya manresulta ed sobran paka-express na pigaran growth factor odino inactivation na tumor suppressor genes.
Sarayan mutation so manresulta ed ag-ondadakel a selula, a manresulta ed pakawalaay tumor.
Legan ya ombabaleg so tumor, onkayat ed asingger iran tissue tan organ, singa say bladder, rectum, tan saray lymph node.
Diad arum a kaso, saray selula na kanser et onsian ed primeron tumor tan onkayat ed arum a parte na laman panamegley na dala odino lymphatic system, sakey a proseso a tatawagen a metastasis.
No kinmayat lay kanser, mairap lan tambalen.
Say prostate cancer et nayarin naapektoan met na hormonal factors, lalo lay androgen hormone ya testosterone.
Say testosterone so mamapabiskeg ed ibubulaslas na saray selula na prostate cancer, tan dakel a panagtambal ed prostate cancer so mangiyaalibansa na pakabawas ed sayan hormone odino pangamper ed epekto na satan.
Bilang sumaryo, say pathophysiology na prostate cancer et mangilalanor ed agnakontrol ya ibaleg tan pambabaleg na selula ed prostate gland, a nayarin naimpluensyaan na genetic, hormonal, tan environmental factors.
Importante so pakatalos ed saray manunan paraan na sakit pian napawala iray epektibon panagtambal tan napaaligwas so pansumpalan na saray pasyente ya walay prostate cancer.
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Kohli M, Qin R, Jimenez R, Dehm SM: Biomarker-based targeting of the androgen-androgen receptor axis in advanced prostate cancer. Adv Urol. 2012, 2012 (): 781459.
Nelson JB, Hedican SP, George DJ, Reddi AH, Piantadosi S, Eisenberger MA, Simons JW: Identification of endothelin-1 in the pathophysiology of metastatic adenocarcinoma of the prostate. Nat Med. 1995, 1 (9): 944-9.
Msaouel P, Nandikolla G, Pneumaticos SG, Koutsilieris M: Bone microenvironment-targeted manipulations for the treatment of osteoblastic metastasis in castration-resistant prostate cancer. Expert Opin Investig Drugs. 2013, 22 (11): 1385-400.
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What is pathophysiology of prostate cancer?
The pathophysiology of prostate cancer refers to the underlying mechanisms and processes that lead to the development and progression of the disease.
Prostate cancer is a malignant tumor that arises from the cells of the prostate gland, which is a small, walnut-shaped organ located below the bladder in men.
The prostate gland produces seminal fluid, which nourishes and transports sperm.
The exact cause of prostate cancer is not fully understood, but several factors are known to increase the risk of developing the disease.
These include age, family history, race, and certain genetic mutations.
Prostate cancer is more common in older men, with the majority of cases occurring in men over the age of 65.
Additionally, men with a family history of prostate cancer are at an increased risk, as are African American men and men of Caribbean descent.
The pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland.
This can occur due to genetic mutations that lead to the overexpression of certain growth factors or the inactivation of tumor suppressor genes.
These mutations can result in the unregulated growth of cells, leading to the formation of a tumor.
As the tumor grows, it can invade nearby tissues and organs, such as the bladder, rectum, and nearby lymph nodes.
In some cases, cancer cells can break away from the primary tumor and spread to other parts of the body through the bloodstream or lymphatic system, a process known as metastasis.
Once the cancer has spread, it can be more difficult to treat.
Prostate cancer can also be influenced by hormonal factors, particularly the androgen hormone testosterone.
Testosterone can stimulate the growth of prostate cancer cells, and many treatments for prostate cancer aim to reduce the levels of this hormone or block its effects.
In summary, the pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland, which can be influenced by genetic, hormonal, and environmental factors.
Understanding the underlying mechanisms of the disease is crucial for developing effective treatments and improving outcomes for patients with prostate cancer.
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