Patofisiologia di diabétis ta referí na e estudio di e prosesonan fisiológiko desordená ku ta kondusí na desaroyo di diabétis.
Diabetes mellitus ta un grupo di malesanan metabóliko karakterisá pa nivelnan haltu di suku den sanger (glukosa) ku ta resultado di defekto den sekreshon di insulina, akshon di insulina òf tur dos.
E fisiopatologia di diabétis ta enbolbí e interakshon kompleho di faktornan genétiko, ambiental i estilo di bida ku ta kontribuí na desaroyo di e malesa.
Den diabétis tipo 1, e fisiopatologia ta enserá un destrukshon autoimune di e sèlnan beta ku ta produsí insulina den pankreas, loke ta kondusí na un defisiensia den produkshon di insulina.
Esaki ta resultá den un inkapasidat pa regulá e nivel di glukosa den sanger, loke ta kondusí na hiperglikemia (sukre haltu den sanger).
Den tipo 2 di diabétis, e fisiopatologia ta mas kompliká i ta enbolbí tantu resistensia na insulina komo sekretashon di insulina.
Resistensia na insulina ta ora e sèlnan di e kurpa no ta reakshoná korektamente riba insulina, loke ta hiba na un inkapasidat pa usa glukosa pa energia.
Esaki ta pone ku e pankreas ta produsí mas insulina pa purba vense e resistensia, pero despues di tempu e pankreas no por sigui ku e demanda, loke ta kondusí na un bahada den e produkshon di insulina.
Diabétis gestacional, ku ta surgi durante embaraso, ta kousá pa kambionan hormonal ku ta kondusí na resistensia na insulina.
E placenta ta produsí hormona ku por blòkia e akshon di insulina, loke ta hiba na un oumento di e nivel di glukosa den sanger.
E fisiopatologia di diabetes tambe ta envolvi e desaroyo di complicacionnan, manera daño na e vasonan sanguineo, e nervio y e organonan, cu por conduci na problemanan serio di salud manera enfermedad di curason, stroke, enfermedad di riñon y perdida di bista.
Komprondé e fisiopatologia di diabétis ta krusial pa desaroyá tratamentunan efektivo i strategianan di prevenshon pa e malesa króniko aki.
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Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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