Kay cáncer de mama patología fisiológica nisqaqa, kay cáncer de mama rikhurimuyninpi, ñawparisqanpi ima, procesos fisiológicos normales nisqapi, mecanismos nisqapi ima, mana allin kaqkunata rikuchin.
Kayqa, kay genética, epigenética, y factores ambientales kaqkuna, kaykunataq, kay cáncer de mama qallariyninman, yapakuyninman, ñawpaqman puriyninman ima yanapanku.
Kaykunataq mama unquypi aswan riqsisqa:
1. Mutantes genéticos: Wakin genespi mutantes kasqankurayku, kay BRCA1 y BRCA2 genespi, cáncer de mama unquyqa astawan yapakun.
Chay mutacionesqa, herenciajina chayri mana herenciajina kanman.
2. Hormonas mana allinchu kasqanku: Hormonas, astawan estrogeno nisqa, cáncer de mama rikhurinapaq, wiñananpaq ima, yanapakun.
Chay hormonas mana allinchu kasqanrayku chayri estrogeno nisqa hormonas astawan yapakusqanrayku, cáncer rikhurinman.
3. Inflamación: Ñuñuspi juk chʼaki unquy rikhuriptinqa, chay unquyqa astawan yapakun.
Underwood SM: Breast cancer in African American women: nursing essentials. ABNF J. , 17 (1): 3-14.
Paliwal SR, Paliwal R, Agrawal GP, Vyas SP: Liposomal nanomedicine for breast cancer therapy. Nanomedicine (Lond). 2011, 6 (6): 1085-100.
Rauch GM, Adrada BE: Comparison of Breast MR Imaging with Molecular Breast Imaging in Breast Cancer Screening, Diagnosis, Staging, and Treatment Response Evaluation. Magn Reson Imaging Clin N Am. 2018, 26 (2): 273-280.
Wei H, Wang H, Ji Q, Sun J, Tao L, Zhou X: NRBP1 is downregulated in breast cancer and NRBP1 overexpression inhibits cancer cell proliferation through Wnt/β-catenin signaling pathway. Onco Targets Ther. 2015, 8 (): 3721-30.
Eden JA: Human breast cancer stem cells and sex hormones--a narrative review. Menopause. 2010, 17 (4): 801-10.
Schneider BP, Winer EP, Foulkes WD, Garber J, Perou CM, Richardson A, Sledge GW, Carey LA: Triple-negative breast cancer: risk factors to potential targets. Clin Cancer Res. 2008, 14 (24): 8010-8.
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Pathophysiology of breast cancer refers to the abnormal changes in the normal physiological processes and mechanisms that occur in the development and progression of breast cancer.
It involves the study of the underlying biological and molecular mechanisms that lead to the uncontrolled growth and spread of cancer cells in the breast tissue.
This includes the genetic, epigenetic, and environmental factors that contribute to the initiation, promotion, and progression of breast cancer.
Some of the key pathophysiological changes in breast cancer include:
1. Genetic mutations: Mutations in certain genes, such as BRCA1 and BRCA2, can increase the risk of developing breast cancer.
These mutations can be inherited or acquired during a person's lifetime.
2. Hormonal imbalances: Hormones, particularly estrogen, can play a role in the development and growth of breast cancer.
An imbalance in hormone levels or exposure to excess estrogen can increase the risk of breast cancer.
3. Inflammation: Chronic inflammation in the breast tissue can contribute to the development of breast cancer by promoting the growth and survival of cancer cells.
4. Tumor microenvironment: The environment surrounding the tumor, including the extracellular matrix, immune cells, and blood vessels, can influence the growth and spread of breast cancer.
5. Metastasis: Breast cancer can spread to other parts of the body through the lymphatic system or bloodstream, leading to the formation of secondary tumors in distant organs.
Understanding the pathophysiology of breast cancer is crucial for developing effective prevention, early detection, and treatment strategies.
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