Cudurka wadnaha, oo sidoo kale loo yaqaano myocardial infarction, wuxuu ku lug leeyahay carqaladeynta qulqulka dhiigga ee qayb ka mid ah muruqyada wadnaha, taasoo keenta dhimashada unugyada wadnaha.
Tani waxay caadi ahaan u dhacdaa sababo la xiriira xannibaadda xididdada dhiigga ee xididdada dhiigga, taas oo inta badan ay sabab u tahay atherosclerosis, oo ah xaalad ay xididdada dhiigga ku urursadaan xididdada dhiigga.
Dhirtu waxay ka kooban tahay kolestarool, dufan, waxyaabaha unugyada ka soo baxa, kalsiyum, iyo fibrin.
Marka uu boodhku dillaacayo, wuxuu sababi karaa in dhiig uu istaago, taas oo xannibi karta xididdada dhiigga waxayna ka hor istaagi kartaa in dhiigga oksijiinta leh uu gaaro muruqa wadnaha.
Oxygen la'aantu waxay keentaa in unugyada muruqyada wadnaha ay dhintaan, taasoo keenta wadne xanuun.
Xaddiga waxyeeladu waxay ku xiran tahay cabirka aagga uu dhiiggu ka soo baxo iyo waqtiga u dhexeeya weerarka iyo daaweynta.
Calaamadaha wadnaha waxaa ka mid noqon kara xanuun ama raaxo la'aan laabta ah, neefsasho la'aan, qufac, wareer, iyo xanuun gacmaha, qoorta, daamanka, ama dhabarka.
Daaweynta wadne xanuunka sida caadiga ah waxay ku lug leedahay dib u soo celinta qulqulka dhiigga ee muruqa wadnaha sida ugu dhakhsaha badan ee suurtogalka ah, ama daawo ama habab sida angioplasty iyo stenting ama qalliinka wadnaha ee wadnaha.
Waxaa muhiim ah in la ogaado in cudurka wadnaha uu yahay mid aad u adag oo ay ku lug leeyihiin arrimo badan, oo ay ku jiraan hiddo-wadaha, qaab nololeedka, iyo arrimaha deegaanka.
Waxyaabaha halista u ah wadne xanuunka waxaa ka mid ah cadaadiska dhiigga oo sarreeya, kolestaroolka oo sarreeya, sigaar cabista, sonkorowga, buurnaanta, dhaqdhaqaaq la'aanta jirka, iyo taariikhda qoyska ee cudurka wadnaha.
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Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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Haddii aadan ku darin dhammaan macluumaadka kor ku xusan waxay keeni kartaa dib u dhac ku yimaada ka baaraandegista cabashadaada.
Xiriirka
Fadlan noogu soo dir emayl su'aal kasta / soo jeedin.
What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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