Pathophysiology of lung cancer waxaa loola jeedaa isbedelada ku dhaca geeddi-socodka caadiga ah ee jirka iyo hababka dhaca horumarinta iyo horumarka kansarka sanbabada.
Kansarka sanbabada waa cudur adag oo ka dhasha koritaanka aan la xakamayn karin iyo kala qaybsanaanta unugyada aan caadiga ahayn ee sanbabada.
Unugyadani waxay abuuri karaan burooyin waxayna ku faafi karaan qaybaha kale ee jirka, taasoo keenta calaamado iyo dhibaatooyin kala duwan.
Pathophysiology-ka kansarka sanbabada waxaa ku lug leh dhowr arrimood, oo ay ku jiraan isbeddellada hiddo-wadaha, arrimaha deegaanka, iyo xulashada qaab nololeedka.
Isbedelada hiddo-wadaha waxay ka dhici karaan DNA-da unugyada sanbabada, taasoo keenta koritaanka unugyada iyo kala qaybsanaanta unugyada oo aan la xakamayn.
Isbedeladaas waxaa la dhaxli karaa ama la heli karaa, waxaana keeni kara waxyaabaha keena kansarka, sida qiiqa tubaakada, radon, asbestos, iyo wasakheynta hawada.
Kansarka sanbabada waxaa loo qaybin karaa laba nooc oo waaweyn: kansarka sanbabada ee unugyada yaryar (SCLC) iyo kansarka sanbabada ee unugyada yar yar (NSCLC). NSCLC waxaa sidoo kale loo qaybiyaa saddex nooc: adenocarcinoma, squamous cell carcinoma, iyo large cell carcinoma.
Pathophysiology-ka noocyada kansarka sanbabada waxay ku kala duwanaan karaan, maadaama ay leeyihiin isbeddello hidde ah oo kala duwan waxayna si kala duwan uga jawaabaan daaweynta.
Pathophysiology-ka kansarka sanbabada wuxuu sidoo kale ku lug leeyahay isdhexgalka u dhexeeya unugyada kansarka iyo unugyada ku xeeran, oo ay ku jiraan nidaamka difaaca.
Unugyada kansarka waxay ka baxsan karaan nidaamka difaaca jirka, taasoo u oggolaaneysa inay koraan oo ay ku faafaan si aan la xakamayn karin.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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Haddii aadan ku darin dhammaan macluumaadka kor ku xusan waxay keeni kartaa dib u dhac ku yimaada ka baaraandegista cabashadaada.
Xiriirka
Fadlan noogu soo dir emayl su'aal kasta / soo jeedin.
What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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