Pathophysiology n colon cancer eqqal fal sistem tan d tikarsiwen n data s eway id efes d efes n colon cancer
Cancer wan colon, s isim net colorectal cancer, eqqal ichikilan n cancer id ezzaran daɣ colon meɣ rectum, eqqal nen ichrutan n colon
Alwaq iyyan t issinta s tiw-at s isim net polyp, addobat ad eqqil cancer daɣ azzaman
Pathophysiology n colon cancer eha iddikud n haratan ajjotnen ahanen genetic mutations, inflammation d haratan n ahinzazaɣ
Genetic mutations addobat ad ijji daɣ DNA n colon cells, eway id efes n cell war n ila akayad d tazunt
Imuttiyan win addoben ad aqqilan n etari meɣ ad awḍen daɣ awadim tolas addoben ad azun nen gène tan ajjotnen ilanen edag daɣ efes n izirwan, tazunt d asuj
Asukni daɣ colon, addobat ad eqqal fal tilɣa chund inflammatory bowel disease, addobat deɣ ad issiwid aharoj n cancer n colon
Asukni n aru addobat ad eway id asiflid n chemical tan wi issiwilanen efes n izirwan d tazunt nasan s addobat nen ad eway efes n cancer
Haratan n ahinzazaɣ chund dumu, tamudre d asiwad n isuj n kimichkal addoben deɣ ad aruwan efes n cancer n colon
Tadhilt tajjarat daɣ aksum azeggaɣ d aksum wi iknanen, almital, t amosat t ahusken hullan n cancer n colon
Haratan iyyad chund azuk, asigar d iban n alxidmat n taɣissa addoben ad awin assahat
Alwaq wa id cancer tan n colon id eɣran, addobat ad iddu s ichikilan ajjotnen, ɣur cancer tan n tazwara n colon har ichikilan n aru s cancer eɣran s ichikilan iyyad n taɣissa
Izirwan win cancer addoben ad awḍen daɣ iɣasan d izirwan n tamazduq tolas addoben ad azun s sistem wan lymphatic meɣ bloodstream s idagan ajjotnen chund liver meɣ afor
Asafar n colon cancer ahan ti iddikud n surgery, chemotherapy d radiation therapy, ilzam s edag d edag n cancer
Asukni n azzaman d asafar aqqalan harat i asiwad n isuj n tumast fal as cancer n colon eqqal a ojjaran addobat asafar alwaq wa id eqqalan daɣ ihandagan net wi azzarnen
Akayad n alwaq iyyan chund colonoscopy addobat ad eɣaf cancer n colon daɣ ihandagan net wi azzarnen alwaq wa id addobat a t ejj
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
['Asukni: medikal']
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['Dǎɣ asikin, neural net wa iknanen tirmawen ən assestan, eqal aśohen aśohat dǎɣ alwaq wa id itawan dǎɣ maɗin ən maɗinan. sund, iddikud ən adinat wi atiwasananen as aśohat atiwasanan.']
['Harkuk arha majrad n doctor inek meɣ n awadim n assexat n tazmert n addinat fal aśahat n aśahat. War t-id-t-igdel majrad n aśahat n awadim meɣ azzaman daɣ aśahat fal as harat awa iktabem fal site wen. Afal as ad t-illa aśahat n aśahat n aśahat, asiwel 911 meɣ aru s ehan n aśahat n aśahat wa oharnen daɣ azzaman. War t-illa majrad n aśahat n awadim d aśahat fal site wen meɣ axadam net. BioMedLib meɣ inaxdiman net meɣ awadim wa iknanen fal site wen, war t-illa majrad, iknan meɣ iknan, fal isalan wi dawanen meɣ axadam net']
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['A fal tazamt s tiditt as isalan meɣ isuj win illanen daɣ asuj n asuj net meɣ alxidmaten iknan alxaq inek n alxaq, addobat ad aɣ-d-tazneḍ (meɣ agent inek) isalan meɣ isuj win ad n-ikkes meɣ ad n-isikkid alxidmat']
['Isalan ilzam ad ittusiknan s eɣaf s email (ikayad edag "Contact" i tansa n email)']
['DMCA t-assirɣas as isalan inek n asiwad n alxaq n alxoriya eha isalan win: (1) almaɣna n alxidmat eha alxaq n alxoriya eqqal harat n asiwad eha alxaq n alxoriya; (2) almaɣna n almaɣna eha alxoriya d isalan ajjotnen ijjan aɣ ad n afhaman almaɣna; (3) isalan n alɣisab inek, ahan tansa, amaṭṭaf n tilifun d tansa n email inek; (4) almaɣna inek n as ilan tumast n alxaq n alxoriya as almaɣna wa s eha alxaq eha alxaq eha alxaq war eha alxaq n alxaq eha alxoriya meɣ amatikki net meɣ s alxidmat n achareɣa fuk']
['(5) almaɣna n awen, iktab daɣ azruf n alkad n tiditt, as isalan win isalan aqqalan tiditt tolas ilan tanaya n as artay alxaqqan win alxakum wi s allaɣen as ikna']
['Dǎɣ awa, əmik ən əmik ən alxaq meɣ awadim ən alxaq ən alxaq meɣ awadim atiwafan ye igi ən alxaq ən alxaq.']
['A fal war t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id']
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What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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