Akoma mu yare a wɔfrɛ no myocardial infarction no de mogya a ɛkɔ koma mu ntini no fã bi mu ba ma ekunkum komayare nkwammoaa no.
Nea ɛma eyi ba ne sɛ mogya a akeka bom na esiw mogya ntini a ɛwɔ wo ntini mu kwan, na mpɛn pii no ɛno na ɛma mogya ntini a ɛwɔ wo ntini mu no yɛ kɛse.
Nneɛma a ɛka bom yɛ nkwammoaa mu ntokuru no ne cholesterol, srade, nneɛma a nkwammoaa no gyae yɛ, calcium, ne fibrin.
Sɛ ntokuru bi paapae a, ebetumi ama mogya atɔre agu, na ebetumi asiw mogya ntini no kwan ma mogya a oxygen wom no antumi ankɔ koma no mu.
Saa mframa a entumi nkɔ nnipa mu yi ma komayare no ba.
Sɛnea yare no mu yɛ den no gyina beae a mogya no fa so no ne bere tenten a edi fi bere a yare no bae kosi bere a wɔsa no yare no so.
Sɛ obi nya komayare a, nea ɛma ohu ne yaw anaa ne koko nyɛ no dɛ, ne home yɛ den, ɔfe, ne ti yɛ no totɔtotɔ, ne ne nsa, ne kɔn, n'apantan, anaa ne akyi yɛ no yaw.
Sɛ obi nya komayare a, nea wɔtaa yɛ ne sɛ wɔbɛma mogya no asan akɔ ne koma mu ntɛmntɛm sɛnea wobetumi, denam nnuru a wɔde di dwuma anaa akwan a wɔfa so yɛ oprehyɛn te sɛ angioplasty ne stenting anaa oprehyɛn a wɔde twa koma no ntini mu.
Ɛho hia sɛ yɛhyɛ no nsow sɛ komayare a ɛma obi nya komayare no yɛ nea ɛkyere adwene na nneɛma pii ka ho, a nea efi awo mu, asetra kwan, ne nneɛma a atwa yɛn ho ahyia ka ho.
Nneɛma a ɛma obi nya komayare no bi ne mogya mmoroso, mogya mu srade a ɛdɔɔso, sigaretnom, asikreyare, kɛseyɛ mmoroso, apɔwmuteɛteɛ a obi ntaa nyɛ, ne komayare a obi abusuafo anya.
Saa nneɛma yi a ebetumi ama obi anya komayare no a wobedi so no betumi aboa ma woatumi atew so.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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['DMCA hwehwɛ sɛ wo amanneɛbɔ a ɛfa nea wɔkyerɛ sɛ ɛyɛ mmara a wobu so ho no de nsɛm a edidi so yi ka ho: (1) adwuma a mmara bɔ ho ban a wɔkyerɛ sɛ wɔadi so no ho asɛm; (2) nsɛm a wɔkyerɛ sɛ wɔadi so no ho asɛm ne nsɛm a ɛbɛboa yɛn ma yɛahu baabi a ɛwɔ; (3) wo ho nsɛm a yɛde bedi nkitaho, a wo address, telefon nɔma ne email address ka ho; (4) wo nsɛm a ɛkyerɛ sɛ wugye di sɛ nea wɔabɔ ho sobo no nyɛ nea mmara ma ho kwan; ']
['(5) wo nsahyɛ a ɛkyerɛ sɛ nsɛm a ɛwɔ amanneɛbɔ no mu yɛ nokware, na wowɔ tumi sɛ wode nea wɔkyerɛ sɛ woadi ho dwuma no bedi dwuma; ']
['ne (6) nea ɔwɔ tumi sɛ ɔyɛ biribi ma obi a ɔwɔ tumi sɛ ɔyɛ biribi ma no no nsaano nkyerɛwee.']
['Sɛ woamfa nsɛm a yɛaka yi nyinaa anka ho a, ebetumi ama wo ka no akyɛ.']
['Nkitahodi']
['Yɛsrɛ wo, fa nsɛmmisa anaa nyansahyɛ biara a wowɔ brɛ yɛn wɔ e-mail so.']
What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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