What is pathophysiology of Depression?

Ushbu sahifani tinglang

Depressiyaning patofiziologiyasi nima?

Depressiyaning patofiziologiyasi depressiyaning rivojlanishi va rivojlanishi uchun mas'ul bo'lgan tartibsiz fiziologik jarayonlar yoki mexanizmlarni o'rganishni anglatadi.

Bu depressiya alomatlarining paydo bo'lishiga va davom etishiga sabab bo'ladigan biologik, nevrologik va psixologik omillarni tushunishni o'z ichiga oladi.

Tadqiqotchilar depressiyaga sabab bo'lishi mumkin bo'lgan bir nechta sabablar va mexanizmlarni aniqladilar, shu jumladan genetik omillar, neyrotransmitterlar darajasi va faolligidagi o'zgarishlar, miyaning tuzilishi va funktsiyasidagi o'zgarishlar va atrof-muhitdagi stress omillari.

Depressiyaning patofiziologiyasi murakkab va ko'p qirrali bo'lib, ushbu omillar kombinatsiyasi turli shaxslarda bu kasallikning rivojlanishiga hissa qo'shishi mumkin.

Ma'lumotlar

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Lai CH: The neural markers of MRI to differentiate depression and panic disorder. Prog Neuropsychopharmacol Biol Psychiatry. 2019, 91 (): 72-78.

Robinson RG, Chemerinski E, Jorge R: Pathophysiology of secondary depressions in the elderly. J Geriatr Psychiatry Neurol. 1999, 12 (3): 128-36.

Krishnan KR, McDonald WM, Doraiswamy PM, Tupler LA, Husain M, Boyko OB, Figiel GS, Ellinwood EH: Neuroanatomical substrates of depression in the elderly. Eur Arch Psychiatry Clin Neurosci. 1993, 243 (1): 41-6.

Olsen TS: Pathophysiology of the migraine aura: the spreading depression theory. Brain. 1995, 118 ( Pt 1) (): 307-8.

Normann C: Towards a new model for cellular pathophysiology in affective disorder. Acta Neuropsychiatr. 2000, 12 (3): 77-80.

Adler UC, Marques AH, Calil HM: Inflammatory aspects of depression. Inflamm Allergy Drug Targets. 2008, 7 (1): 19-23.

Rihmer Z, Seregi K, Rihmer A: Parkinson's disease and depression. Neuropsychopharmacol Hung. 2004, 6 (2): 82-5.

Mas'uliyatdan voz kechish: tibbiy

Ushbu veb-sayt faqat ta'lim va axborot maqsadlari uchun mo'ljallangan bo'lib, tibbiy maslahat yoki professional xizmatlar ko'rsatmaydi.

Ma'lumotlardan sog'liqni saqlash muammolari yoki kasalliklarni tashxislash yoki davolash uchun foydalanmaslik kerak va shaxsiy tibbiy maslahat so'raganlar litsenziyaga ega bo'lgan shifokor bilan maslahatlashishlari kerak.

Iltimos, savollarga javoblarni ishlab chiqaradigan neyron tarmog'i, ayniqsa, raqamli tarkibga kelganda noto'g'ri ekanligiga e'tibor bering. Masalan, ma'lum bir kasallik bilan kasallangan odamlar soni.

Har doim shifokoringiz yoki boshqa malakali sog'liqni saqlash provayderining maslahatini so'rang. Hech qachon professional tibbiy maslahatni e'tiborsiz qoldirmang yoki ushbu veb-saytda o'qiganingiz sababli uni so'rashni kechiktirmang. Agar siz tibbiy favqulodda vaziyatga duchor bo'lishingiz mumkin deb o'ylasangiz, darhol 911 ga qo'ng'iroq qiling yoki eng yaqin favqulodda vaziyatlar bo'limiga boring. Ushbu veb-sayt yoki uning ishlatilishi bilan hech qanday shifokor- bemor munosabatlari yaratilmaydi. BioMedLib ham, uning xodimlari ham, ushbu veb-saytga hech qanday hissa qo'shuvchi, bu erda taqdim etilgan ma'lumot yoki uning ishlatilishi bilan bog'liq hech qanday bayonot bermaydi.

Mas'uliyatdan voz kechish: mualliflik huquqi

1998-yilgi raqamli ming yillik mualliflik huquqi to'g'risidagi qonun, 17 U.S.C. 512-moddasi (DMCA) Internetda paydo bo'lgan materiallar AQSh mualliflik huquqi to'g'risidagi qonun bo'yicha o'z huquqlarini buzadi deb hisoblaydigan mualliflik huquqi egalari uchun choralar ko'rsatadi.

Agar siz bizning veb-saytimiz yoki xizmatlarimiz bilan bog'liq bo'lgan har qanday tarkib yoki material sizning mualliflik huquqingizni buzadi deb yaxshi ishonchga ega bo'lsangiz, siz (yoki sizning vakilingiz) bizga tarkib yoki materialni olib tashlashni yoki unga kirishni to'xtatishni so'rab xabar yuborishingiz mumkin.

Xabarlar yozma ravishda elektron pochta orqali yuborilishi kerak (elektron pochta manzili uchun "Muloqot" bo'limiga qarang).

DMCA sizning da'vo qilingan mualliflik huquqi buzilganligi to'g'risidagi xabarnomangizda quyidagi ma'lumotlarni o'z ichiga olishini talab qiladi: (1) da'vo qilingan mualliflik huquqi buzilgan asarning tavsifi; (2) da'vo qilingan mualliflik huquqi buzilgan tarkibning tavsifi va bizga tarkibni topishga imkon beradigan etarli ma'lumotlar; (3) siz uchun aloqa ma'lumotlari, shu jumladan sizning manzilingiz, telefon raqami va elektron pochta manzili; (4) siz tomonidan da'vo qilingan tarzda tarkib mualliflik huquqi egasi yoki uning vakili yoki har qanday qonun tomonidan ruxsat berilmaganligiga ishonchingiz borligi to'g'risidagi bayonot;

(5) siz tomonidan yolg'on guvohlik berish jazosi ostida imzolangan, bildirishnomadagi ma'lumotlar to'g'ri ekanligi va siz buzilgan deb da'vo qilingan mualliflik huquqlarini amalga oshirish vakolatiga ega ekanligingiz to'g'risidagi bayonot;

va (6) mualliflik huquqi egasining yoki mualliflik huquqi egasi nomidan harakat qilishga vakolatli shaxsning jismoniy yoki elektron imzosi.

Yuqoridagi barcha ma'lumotlarni kiritmaslik sizning shikoyatingizni ko'rib chiqishni kechiktirishi mumkin.

Aloqa qilish

Iltimos, har qanday savol / taklif bilan bizga elektron pochta xabarini yuboring.

What is pathophysiology of depression?

Pathophysiology of depression refers to the study of the disordered physiological processes or mechanisms that are responsible for the development and progression of depression.

It involves understanding the underlying biological, neurological, and psychological factors that contribute to the onset and persistence of depressive symptoms.

Researchers have identified several potential causes and mechanisms that may contribute to depression, including genetic factors, alterations in neurotransmitter levels and activity, changes in brain structure and function, and environmental stressors.

The pathophysiology of depression is complex and multifaceted, and it is likely that a combination of these factors contributes to the development of the disorder in different individuals.

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