Jàngoroy feebar bi (pathophysiologie) mooy gëstuy doxaliinu yaram yi di jaxase ngir jur feebar bi.
Diabète mellitus mooy xeetu feebar buy tax nit di am suukar su bari ci deret ji (glucose) ndax ay gàkk ci suuxu insulin, walla ci jëfu insulin, walla ci ñoom ñaar.
Jàngoroy sukkandikoo ci yaram dafay ànd ak ay wàll yu jafe ci wàllu gene, ci wàllu mbay ak ci wàllu dund, loolu lépp a waral jàngoro ji.
Ci feebar bi di diabetes bu 1eelu xeetu feebar bi, pathophysiologie bi dafay indi ag yàqute gu auto-immune ci selil beta yiy jur insulin ci pancreas bi, loolu di indi ag ñàkk ci wàllu jur insulin.
Loolu dafay tax ba mënatul a saytu tolluwaayu suukar ci deret ji, di indi jàngoroy suukar bu bare ci deret ji (hyperglycémie).
Ci feebar bi di type 2 diabetes, pathophysiology bi dafa gën a jafe te dafay indi insulin resistance ak insulin secretion bu yàqu.
Jëfekaay biy saytu insulin dafay am su ay selilóor ci yaram yi nanguwul insulin bu baax, di tax ñu mënatul jëfandikoo glucose ngir sàkku doole.
Loolu moo tax ba bànqaasu yaram di génne insulin bu ëpp ngir jéem a jéggi ndànk-ndànk bi, waaye ci ginnaaw jamono, bànqaasu yaram mënul a doyloo soxla ji, loolu di indi wàññi gu insulin.
Diabète bu ëmb bi, mi ngi am ci biir ëmb, dafay juddoo ci soppi hormon yi indi ag bañ a nangu insulin.
Xale bi dafa am ay ormon yu mën a tere insulin di jëf, loolu di indi yokkute ci suuxu deret ji.
Jàngoroy suukar dafay jur ay jafe-jafe, niki yàq deret ji, ay yax ak ay cér, loolu mën na indi ay feebar yu metti niki feebarub xol, ay jàdd, ay feebarub tëgg ak ñàkk gis.
Xam-xamu pathophysiologie bu jàngoroy suukar am na solo lool ngir mën a sos ay faj yu am njariñ ak ay pexe yu ñu mën a aar ci jàngoro ju yàgg jii.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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