Jàngoroy xol bu ñuy wax infarctus du myocarde dafay indi ag yàqute ci doxug deret ji jëm ci wàll ci yaram wi, di indi deewu ay selilëer ci xol bi.
Li ko waral mooy ne, deret ji dafa tëj buumu deret ji, loolu nag, li ko waral mooy aterosikleroos, di jàngoro ji tax ba ay deret di dajaloo ci biir buumu der yi.
Xobam dafa am ay kolesterol, ay diw, ay cell yuy yàq der, ay calcium ak ay fibrin.
Bu ñu ko defee, deret ji day suux, di tëj deret ji, di tere deret ji am oksijenn ba àgg ci xol bi.
ñàkkum oksijiin gii moo tax ay selil yu mbëj di dee, indi fitna ci xol bi.
Xàll-jàll bi mu jur day aju ci yaatuwaay bi ko artère bi tëj di joxe ak diir bi dox diggante bi ko jàkka ji dal ak bi ñu koy faj.
Ay firndey jàngoroy xol mën nañ indi metit ci dënn walla coono, nelaw bu gàtt, tiitaange, yëg-yëg, metit ci loxo, loos, bëñ walla gannaaw.
Jëfandikoo ag wallu ag dalub xol dafay tax deret ji dellu dox ci xol bi ci lu gaaw, ci ay doktoor walla ay doxalin yu mel ni angioplasti ak stenting walla bypass bu artère coronaire.
Li am solo mooy xam ne pathophysiologie bu jàngoroy xol dafa jafe te dafa ëmb ay fànn yu bari, lu ci mel ne gene, dund ak li wër yaram.
Ñi mën a jur ag jàdd ci xol dañuy bokk ci ñi am feebar bu kawe ci deret, kolesterol bu kawe, saxar, jàngoroy sukk, tàngoor, ñàkk jëflante ak njaboot gu am jàngoroy xol.
Jëfandikoo yëf yooyu mën na wàññi sa jafe-jafey am ag jàppalekatu xol.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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['Danga war a wutal sa doktoor walla beneen fajkat bu am xam-xam ci lu jëm ci wér-gu-yaram. Bul sàggane walla nga gaaw a wutal sa doktoor ndax dara lu nga jàng ci dal bii. Soo xalaatee ne am nga lu la soxla ci wér-gu-yaram, wool 911 walla nga dem ci fajukaay bu la gën a jege ci saa si. Dal bii walla jëfandikoo ko taxul nga nekk ak ab doktoor walla ab jarag. BioMedLib walla ay liggéeykatam, walla kenn ci ñi koy jëfandikoo, duñu wax dara, muy lu leer mbaa lu leeradi, ci lu jëm ci xibaar yi ñu leen di jox fii walla ci ni ñu koy jëfandikoo.']
['Séddo: sañ-sañu jëfandikoo']
['Sàrtu sàmm-sañu-xët yi ñu bind ci Internet ci atum 1998 (Digital Millennium Copyright Act of 1998), 17 U.S.C. § 512 (ci angale mooy DMCA) dafay may boroom-sañu-xët yi ñu jàpp ne ay mbind yu feeñ ci Internet dañuy yàq seen sañ-sañ ci yoon wi ñu bind ci Amerig. ']
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['Bind nañu ay yëgle ci mbind, ci ab bataaxal (Xoolal "Contact" ngir xam màkkaanu bataaxal bi).']
['DMCA dafa digle ne sa bataaxal bu jëm ci jàddug sañ-sañ bu ñu sos war na ëmb li ci topp: (1) xët wu jëm ci liggéey bi ñu sos ne jàdd nañ ko; (2) xët wu jëm ci li ñu sos ne jàdd nañ ko ak ay xibaar yu doy ngir may nu nu nu man a gis li mu ëmb; (3) ay xibaar yu jëm ci yaw, boole ci sa màkkaanu dal, sa limu telefóni ak sa màkkaanu imeel; (4) ab kàddu bu jóge ci yaw bu lay xamal ne am nga yaakaar bu wér ne li nga sos ci anam wi ñu la ko sosu, moom boroom sañ-sañ bi, walla ki ko dénk, walla benn yoon, nanguwu ko; ']
['(5) ab bataaxal bu ñu la jox, nga dëggal ci sa loxo ne li nga bind dëgg la te am nga sañ-sañu sàmm sañ-sañu jëfandikoo sañ-sañu bind bi ñu la sosal ne yàqu na;']
['ak (6) benn màndarga buy firndeel walla buy wone ay màndarga yuy wone ne moom la sañ-sañu jëfandikoo walla mu ngi koy jëfandikoo ci turu moom. ']
['Suñ la ci dugalul lépp lu ñu wax ci kaw, mën na tax ba say tawat di gaaw a jàppale.']
['Waxtaan']
['Yónneel nu ab imeel bu la laaj walla nga am ay xalaat.']
What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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['Nu tàmbali ci 35 milyoŋi téerey xam-xamu jëmm ci PubMed/Medline. Te itam ci xëti web yi ci RefinedWeb.']
