Jàngoroy sëqëtukaay dafay tekki ay jëfekaay ak ay doxalin yuy tax jàngoro ji di law.
Kanser prostate ab jàngoro la buy juddoo ci ay selilëer yu nekk ci prostate, di ab cér bu ndaw bu am melow mbalit bu nekk ci suufu gémmiñ ci góor.
Mbëjfeppal bi dafay jur ndoxum gerte, muy dundal ak di yóbbu gerte gi.
Li waral jàngoroy prostate bi leeragu ko ba noppi, waaye am na lu bari lu ñu xam ne moo yokk wàññi-gàddaay bi.
Ñoo ngi ci bokk: mag, njaboot, xeetu nit ak yenn xeetu gene.
Kanser prostate mi ngi gën a bare ci góor ñi gën a màggat, te li ëpp ci nit ñi am kansere mooy ñi am 65 at.
Te itam, góor ñi seen njaboot am jàngoroy prostate ñoo gën a am jafe-jafe bi, niki góor ñi dëkk Afrig, Amerig ak waa Kariibi.
Jàngoroy sëqëtukaay dafay jur yokkute ak séddale ay selil yu amul benn yoon ci biir sëqëtukaay bi.
Loolu mën na am ndax ay coppite yuy tax ñu gën a jëfandikoo ay wàll yuy yokk walla ñu bañ a jëfandikoo ay geney supresseurs de tumeur.
Yëngu-yëngu yooyu mën na tax ay selil di yokku buñ mën a saytu, ba tax ñu jur ab sëq.
Bu màndarga ji di màgg, mën na song yaram yi ko wër ak ay cér yu ko wër, niki lëj-lëj bi, tëgg bi ak ay nosukaay yu jege.
Ci yenn mbir yi, ay selilëer yuy law mën nañu tàqale ak màndargam yaram wi ñu ko jëkka def te law ci yeneen wàll yi ci yaram wi jaare ko ci deret wi walla ci sistem bu limfa bi, loolu ñu naan metastaas.
Bu jàngoro ji tasaaroo, dina jafe ci faj gi.
Kanser prostate mën na ame ay wàll ci wàllu hormones, rawatina ci wàllu hormones androgènes yi, testosterone.
Testosterone mën na yokk màggug selil prostate cancer, te ay faj yu bari ci prostate cancer dañuy fexee wàññi tolluwaayu hormone boobu walla ñu tere ay jëfam.
Ci gàttal, pathophysiologie bu jàngoroy prostate dafay jur yokkute ak séddale ay selil yu amul benn yoon ci biir prostate bi, te geneen wàll, hormones ak lu ko wër mën na ko indi.
Xam-xamu doxalin yi waral jàngoro ji am na solo lool ngir mën a sos ay faj yu am njariñ te gën a wér ci ñi am jàngoroy prostate.
Zobniw CM, Causebrook A, Fong MK: Clinical use of abiraterone in the treatment of metastatic castration-resistant prostate cancer. Res Rep Urol. 2014, 6 (): 97-105.
Lim HY, Agarwal AM, Agarwal N, Ward JH: Recurrent epistaxis as a presenting sign of androgen-sensitive metastatic prostate cancer. Singapore Med J. 2009, 50 (5): e178-80.
Kohli M, Qin R, Jimenez R, Dehm SM: Biomarker-based targeting of the androgen-androgen receptor axis in advanced prostate cancer. Adv Urol. 2012, 2012 (): 781459.
Nelson JB, Hedican SP, George DJ, Reddi AH, Piantadosi S, Eisenberger MA, Simons JW: Identification of endothelin-1 in the pathophysiology of metastatic adenocarcinoma of the prostate. Nat Med. 1995, 1 (9): 944-9.
Msaouel P, Nandikolla G, Pneumaticos SG, Koutsilieris M: Bone microenvironment-targeted manipulations for the treatment of osteoblastic metastasis in castration-resistant prostate cancer. Expert Opin Investig Drugs. 2013, 22 (11): 1385-400.
Kotani K, Sekine Y, Ishikawa S, Ikpot IZ, Suzuki K, Remaley AT: High-density lipoprotein and prostate cancer: an overview. J Epidemiol. 2013, 23 (5): 313-9.
Jadvar H: Molecular imaging of prostate cancer: a concise synopsis. Mol Imaging. , 8 (2): 56-64.
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['Bind nañu ay yëgle ci mbind, ci ab bataaxal (Xoolal "Contact" ngir xam màkkaanu bataaxal bi).']
['DMCA dafa digle ne sa bataaxal bu jëm ci jàddug sañ-sañ bu ñu sos war na ëmb li ci topp: (1) xët wu jëm ci liggéey bi ñu sos ne jàdd nañ ko; (2) xët wu jëm ci li ñu sos ne jàdd nañ ko ak ay xibaar yu doy ngir may nu nu nu man a gis li mu ëmb; (3) ay xibaar yu jëm ci yaw, boole ci sa màkkaanu dal, sa limu telefóni ak sa màkkaanu imeel; (4) ab kàddu bu jóge ci yaw bu lay xamal ne am nga yaakaar bu wér ne li nga sos ci anam wi ñu la ko sosu, moom boroom sañ-sañ bi, walla ki ko dénk, walla benn yoon, nanguwu ko; ']
['(5) ab bataaxal bu ñu la jox, nga dëggal ci sa loxo ne li nga bind dëgg la te am nga sañ-sañu sàmm sañ-sañu jëfandikoo sañ-sañu bind bi ñu la sosal ne yàqu na;']
['ak (6) benn màndarga buy firndeel walla buy wone ay màndarga yuy wone ne moom la sañ-sañu jëfandikoo walla mu ngi koy jëfandikoo ci turu moom. ']
['Suñ la ci dugalul lépp lu ñu wax ci kaw, mën na tax ba say tawat di gaaw a jàppale.']
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['Yónneel nu ab imeel bu la laaj walla nga am ay xalaat.']
What is pathophysiology of prostate cancer?
The pathophysiology of prostate cancer refers to the underlying mechanisms and processes that lead to the development and progression of the disease.
Prostate cancer is a malignant tumor that arises from the cells of the prostate gland, which is a small, walnut-shaped organ located below the bladder in men.
The prostate gland produces seminal fluid, which nourishes and transports sperm.
The exact cause of prostate cancer is not fully understood, but several factors are known to increase the risk of developing the disease.
These include age, family history, race, and certain genetic mutations.
Prostate cancer is more common in older men, with the majority of cases occurring in men over the age of 65.
Additionally, men with a family history of prostate cancer are at an increased risk, as are African American men and men of Caribbean descent.
The pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland.
This can occur due to genetic mutations that lead to the overexpression of certain growth factors or the inactivation of tumor suppressor genes.
These mutations can result in the unregulated growth of cells, leading to the formation of a tumor.
As the tumor grows, it can invade nearby tissues and organs, such as the bladder, rectum, and nearby lymph nodes.
In some cases, cancer cells can break away from the primary tumor and spread to other parts of the body through the bloodstream or lymphatic system, a process known as metastasis.
Once the cancer has spread, it can be more difficult to treat.
Prostate cancer can also be influenced by hormonal factors, particularly the androgen hormone testosterone.
Testosterone can stimulate the growth of prostate cancer cells, and many treatments for prostate cancer aim to reduce the levels of this hormone or block its effects.
In summary, the pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland, which can be influenced by genetic, hormonal, and environmental factors.
Understanding the underlying mechanisms of the disease is crucial for developing effective treatments and improving outcomes for patients with prostate cancer.
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