Àìsàn Alzheimer jẹ́ àìsàn kan tó ń ṣàkóbá fún ọpọlọ gan-an, ó sì máa ń mú kí àwọn èèyàn pàdánù ìrántí, kí wọ́n má mọ nǹkan dáadáa, ó sì máa ń yí ìwà wọn pa dà.
A ò tíì mọ ohun tó ń fa àrùn Alzheimer ní pàtó, àmọ́ wọ́n gbà gbọ́ pé àwọn nǹkan bíi àbùdá, àyíká àti ọ̀nà ìgbésí ayé ló ń fa àrùn náà.
Lára àwọn nǹkan tó lè fa àrùn Alzheimer ni:
1. Àmì àbùdá: Wọ́n ti ṣàwárí àwọn àbùdá kan tó ń mú kí ewu àrùn Alzheimer pọ̀ sí i, àgàgà àbùdá apolipoprotein E (APOE).
2. Ọ̀dọ́mọdé: Bí èèyàn bá ṣe ń dàgbà sí i, bẹ́ẹ̀ ló máa ń pọ̀ sí i, èyí tó pọ̀ jù lọ lára wọn ló sì máa ń di aláìsàn Alzheimer lẹ́yìn tí wọ́n bá ti pé ọmọ ọdún 65.
3. Ìtàn ìdílé: Tó o bá ti ní àrùn Alzheimer nínú ìdílé rẹ, ó lè mú kí ewu tó wà fún ọ láti ní àrùn náà pọ̀ sí i.
4. Àwọn ọgbẹ́ orí: Àwọn ọgbẹ́ orí tó ti ṣẹlẹ̀ rí, pàápàá àwọn tó máa ń mú kí ẹnì kan dákú, lè mú kí ewu àrùn Alzheimer pọ̀ sí i.
5. Àwọn nǹkan tó ń fa àrùn ọkàn àti ẹ̀jẹ̀: Àwọn àìsàn bíi ẹ̀jẹ̀ tó ga, èròjà cholesterol tó ga, àti àrùn àtọ̀gbẹ lè mú kí ewu àrùn Alzheimer pọ̀ sí i.
6. Àwọn nǹkan tó ń fa ọ̀nà ìgbésí ayé: Ìgbésí ayé tí kò fi bẹ́ẹ̀ rìn, oúnjẹ tí kò dára, àti àìsí nǹkan tó ń mú ọpọlọ ṣiṣẹ́ lè mú kí àrùn Alzheimer pọ̀ sí i.
7. Ìpalára: Ìpalára tí kò lópin nínú ọpọlọ lè mú kí àrùn Alzheimer wáyé.
8. Àníyàn tó ń yọrí sí oxidative stress: Ìyàtọ̀ tó wà láàárín bí ara ṣe ń mú àwọn èròjà tó ń jẹ́ free radicals jáde àti bí ara ṣe ń mú wọn kúrò nínú ara lè fa àrùn Alzheimer.
9. Àwọn àlàfo amyloid àti àwọn àlàfo neurofibrillary: Ìkójọpọ̀ àwọn àlàfo amyloid àti àwọn àlàfo neurofibrillary nínú ọpọlọ jẹ́ àmì àìsàn Alzheimer, ṣùgbọ́n ipa tí wọ́n ń kó nínú ìdàgbàsókè àìsàn náà kò yé gbogbo èèyàn.
Ó ṣe pàtàkì láti kíyè sí i pé ohun tó ń fa àrùn Alzheimer nira gan-an, ó sì ṣeé ṣe kó jẹ́ pé àwọn nǹkan wọ̀nyí ló ń fa àrùn náà.
Ìwádìí ń lọ lọ́wọ́ láti túbọ̀ lóye àwọn ohun tó ń fà á, kí wọ́n sì ṣe ìtọ́jú tó gbéṣẹ́ fún àìsàn burúkú yìí.
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Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
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Ìfọ̀rọ̀wérọ̀
Jọwọ fi imeeli ranṣẹ si wa pẹlu eyikeyi ibeere / imọran.
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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