Àìsàn Alzheimer jẹ́ àìsàn kan tó ń ba ọpọlọ jẹ́, èyí tó sábà máa ń kọ lu àwọn àgbàlagbà.
Bá a ṣe ń darúgbó, bẹ́ẹ̀ ni ewu àìsàn Alzheimer ṣe ń pọ̀ sí i, ó sì sábà máa ń bá àwọn tó ti pé ọmọ ọdún márùn-ún.
Àmọ́ ṣá o, àwọn èèyàn tó ti pé ọmọ ọgbọ̀n ọdún, ogójì ọdún, àti àádọ́ta ọdún lè ní àrùn Alzheimer tó ti bẹ̀rẹ̀ tẹ́lẹ̀, bó tilẹ̀ jẹ́ pé kì í fi bẹ́ẹ̀ wọ́pọ̀.
Àwọn àbùdá tún ń kó ipa pàtàkì nínú bí àrùn Alzheimer ṣe ń yọrí sí.
Àwọn tó ní àrùn náà nínú ìdílé wọn ní ewu tó ga jù lọ láti ní àrùn náà fúnra wọn.
Yàtọ̀ síyẹn, àwọn àyípadà kan tó wà nínú àbùdá, irú bí àbùdá APOE-e4, tún lè mú kí àrùn Alzheimer pọ̀ sí i.
Lára àwọn nǹkan mìíràn tó tún lè fa àrùn Alzheimer ni àwọn tó ti fara pa ní orí rí, àìsàn ọkàn àti ẹ̀jẹ̀, àti àwọn nǹkan tó máa ń mú kéèyàn máa gbé ìgbésí ayé bó ṣe máa ń mu sìgá, bó ṣe máa ń sanra tó, àti bí kò ṣe máa ń ṣe eré ìmárale.
Ó ṣe pàtàkì láti kíyè sí i pé bó tilẹ̀ jẹ́ pé àwọn nǹkan kan lè mú kí ewu àrùn Alzheimer pọ̀ sí i, kì í ṣe ohun tó máa ń ṣẹlẹ̀ nígbà téèyàn bá ń darúgbó, kì í sì í ṣe gbogbo èèyàn tó bá ń darúgbó ló máa ní àrùn náà.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Bakhtiari S, Moghadam NB, Ehsani M, Mortazavi H, Sabour S, Bakhshi M: Can Salivary Acetylcholinesterase be a Diagnostic Biomarker for Alzheimer? J Clin Diagn Res. 2017, 11 (1): ZC58-ZC60.
Ìyàsímímọ́: ìtọ́jú ìlera
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A ò gbọ́dọ̀ lo ìsọfúnni tó wà nínú ìwé náà láti ṣe àyẹ̀wò tàbí láti wo àìsàn tàbí àìsàn kan wò, àwọn tó bá sì ń wá ìmọ̀ràn nípa ìṣègùn fúnra wọn gbọ́dọ̀ bá dókítà tó ní ìwé àṣẹ sọ̀rọ̀.
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Ti o ko ba ṣafikun gbogbo alaye ti o wa loke le ja si idaduro ti ṣiṣe ẹdun rẹ.
Ìfọ̀rọ̀wérọ̀
Jọwọ fi imeeli ranṣẹ si wa pẹlu eyikeyi ibeere / imọran.
Who gets alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects older adults.
The risk of developing Alzheimer's disease increases with age, and it is most common in people over the age of 65.
However, early-onset Alzheimer's can occur in people in their 30s, 40s, and 50s, although this is less common.
Genetics also play a role in the development of Alzheimer's disease.
People with a family history of the disease are at a higher risk of developing it themselves.
Additionally, certain genetic mutations, such as the APOE-e4 gene, can increase the likelihood of developing Alzheimer's.
Other risk factors for Alzheimer's disease include a history of head injuries, cardiovascular disease, and lifestyle factors such as smoking, obesity, and lack of exercise.
It is important to note that while certain factors can increase the risk of developing Alzheimer's disease, it is not a normal part of aging, and not everyone who ages will develop the disease.
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