Patofisiologi kanker paru-paru mengacu bak perubahan proses dan mekanisme fisiologis normal nyang terjadi bak perkembangan dan perkembangan kanker paru-paru.
Kanker paru-paru nyan keuh peunyaket kompleks yang timbul dari pertumbuhan dan pembagian sel abnormal yang hana terkontrol di paru-paru.
Sel-sel nyo jeut membentuk tumor dan menyebar ke bagian laen tuboh, yang jeut keu macam-macam gejala dan komplikasi.
Patofisiologi kanker paru-paru melibatkan beberapa faktor, termasuk mutasi genetik, faktor lingkungan, dan pilihan gaya hidup.
Mutasi genetik jeut terjadi bak DNA sel paru-paru, yang jeut keu pertumbuhan dan pembelahan sel yang hana terkontrol.
Mutasi nyo jeut diwariskan atau didapat, dan jeut disebabkan oleh paparan karsinogen, lage asap tembakau, radon, asbes, dan polusi udara.
Kanker paru jeut diklasifikasikan jeut dua jenis utama: kanker paru sel ubit (SCLC) dan kanker paru sel ken-kecil (NSCLC). NSCLC dibagi lom jeut keu lhee subtipe: adenokarsinoma, karsinoma sel skuamosa, dan karsinoma sel rayek.
Patofisiologi dari jenis kanker paru-paru nyoe jeut berbeda, karena awak nyan na mutasi genetik yang berbeda dan merespon secara berbeda terhadap pengobatan.
Patofisiologi kanker paru-paru cit melibatkan interaksi antara sel kanker ngen jaringan sekitar jih, termasuk sistem kekebalan.
Sel kanker jeut menghindar dari sistem kekebalan, memungkenkan jih tumbuh dan menyebar hana terkontrol.
Seulaen nyan, lingkungan mikro tumor jeut ji promosi pertumbuhan tumor ngon metastasis ngen ji sediakan lingkungan pendukung untuk sel kanker.
Patofisiologi kanker paru-paru nyan proses nyang kompleks ngon dinamis, ngon peneliti terus bekerja untuk leubeh meuphom mekanisme yang mendasari untuk mengembangkan perawatan yang leubeh efektif dan meningkatkan hasil pasien.
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Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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