Pathophysiology of diabetes kyerɛ sɛ yareɛ no yɛ yareɛ a ɛsae nnipa.
Asikreyare yɛ yareɛ a ɛsae nnipa na ɛsan ma mogya mu nneɛma nketenkete bi te sɛ asikre (glucose) yɛ adwuma yiye.
Akwahosan ho nimdeɛ a ɛfa asikreyare ho no fa sɛnea awosu, nneɛma a atwa yɛn ho ahyia, ne asetena mu nneɛma di dwuma ma yare no ba no ho.
Wɔ type 1 asikreyare no mu no, yare no fi nipadua mu nkwammoaa a wͻde siw yare kwan a wͻde siw yare kwan no mu.
Eyi ma wontumi nhyɛ mogya mu asikre no so ma ɛde ba wɔ mogya mu (hyperglycemia).
Wɔ type 2 asikreyare mu no, yareɛ no ho nsɛm mu dɔ na ɛfa insulin ho paneɛ a ɛko tia yareɛ no ne insulin a ɛmma nipadua no ntumi nkeka ne ho.
Insulin a ekura nipadua mu ahoɔden no ba bere a nipadua mu nkwammoaa no ntumi nnyina insulin ano yiye, na ɛma wontumi mfa ahoɔden a ɛwɔ glucose mu no nni dwuma yiye.
Eyi ma ekuru no nya insulin pii de di dwuma de ko tia yare no, nanso bere rekɔ so no, ekuru no ntumi nyɛ adwuma sɛnea ɛsɛ, na ɛma insulin a ɛyɛ no so tew.
Nyinsɛn mu asikreyare a ɛba wɔ nyinsɛn mu no fi nsakrae a ɛba nipadua mu a ɛma nipadua no ntumi nnyina insulin ano no.
Nkwadaa a wɔwo wɔn no nya nkwammoaa a etumi siw insulin kwan ma mogya mu asikre yɛ kɛse.
Yareɛ a ɛde yareɛ ba no de ɔhaw pii ba, te sɛ mogya ntini a ɛhaw adwene, ne nipadua mu akwaa a ɛtumi de yareɛ ba te sɛ komayareɛ, yareɛ a ɛma obi fã dwudwo, asaabo yareɛ, ne anifirafoɔ yareɛ.
Sɛ yɛbɛte yareɛ a ɛde asikreyare ba no ase a, ɛho hia sɛ yɛfa kwan pa so sa yareɛ no na yɛhwɛ sɛ ɛremma obi nnya bi.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
['Abɔdin: ayaresa']
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["Hwehwɛ afotu fi wo dɔkota anaa ayaresafo a wɔfata hɔ bere biara wɔ yareɛ ho. Nnyae ayaresa ho afotuo a wɔn a wɔn ho akokwaw de ma no ho adwenemu anaa twentwɛn wo nan ase sɛ worebɛhwehwɛ esiane biribi a woakenkan wɔ wɛbsaet yi so nti. Sɛ ɛyɛ wo sɛ wowɔ yareɛ ho nsɛmmisa a, frɛ 911 anaa kɔ ayaresabea a ɛbɛn wo pɛɛ ntɛm ara. Saa wɛbsaet yi anaa dwuma a wode di no mma wonnya oduruyɛfo ne ɔyarefo ayɔnkofa biara. BioMedLib anaa n'adwumayɛfoɔ anaa obiara a ɔde ne ho bɔ wɛbsaet yi ho dawuro biara nni mu, a ɛkyerɛ anaa enni mu, fa nsɛm a wɔde ama wɔ ha anaa dwuma a wɔde di no ho."]
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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