Ubulwele bwa ku mutima ubulenga umuntu ukufwa, ubwitwa no kuti myocardial infarction, bulenga umulopa ukuleka ukwingila mu mubili wa muntu, kabili ici cilenga insandesande sha mutima ukufwa.
Ici ilingi cicitika pa mulandu wa kuti umulopa waleka ukukontoka mu mishipa, kabili ilingi ici cilenga ni co umulopa waleka ukukontoka mu mishipa.
Ifi fintu ni cholesterol, ifiko, ifiko ifitwala ifiko ku mubili, calcium, ne fibrin.
Nga ca kuti icilonda calepuka, kuti calenga umulopa ukupangwa, kabili kuti waisalika umulopa no kulesha umulopa wa oxygen ukufika ku mutima.
Ukubulwa kwa oxygen kulenga insandesande sha ku mutima ukufwa, ne ci cilenga umutima ukuleka ukubomba.
Ifyo icilonda caonaika filapusanapusana ukulingana ne ncende yalekabila umulopa mu mulopa walekalipa no nshita yapitilepo ukufuma apo icilonda cabelele ukufika ilyo icilonda calekabila ukucitwa.
Ifishibilo fya bulwele bwa ku mutima kuti fyaba kucululuka nelyo ukufulwa mu cifuba, ukufilwa ukupeema, ukufulwa sana, ukufulungana, no kucululuka mu maboko, mu mukoshi, mu kanwa, nelyo mu numa.
Pa kundapa ubulwele bwa ku mutima ilingi line palapita ukubwesha umulopa mu mishipa ya mutima bwangu bwangu, ukupitila mu miti nelyo ukubomfya inshila pamo nga angioplasty no kubikamo stent nelyo ukulepula umwela wa ku mutima.
Cacindama ukwishiba ukuti ubulwele bwa ku mutima bwalikosa kabili bwalikwata ifilenga, pamo nga ifilenga umuntu ukufyalwa, ifyo umuntu aikala, ne fya mu bwikashi.
Ifingalenga umuntu ukulalwala umutima ni fi: ukulalwala sana umulopa, ukukwata sana cholesterol, ukupeepa, ubulwele bwa shuga, ukufina sana, ukukanabomba sana imilimo ya mubili, no kukwata ubulwele bwa ku mutima mu lupwa.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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