Pathophysiology ya yikola ya shuga yinalumbunuka kulilongesa cha yitanga ya yikola ya mujimba yize yakuneha yikola ya shuga.
Musongo wa chindululu uli chizavu cha yikola ya mujimba yize yakupwa ha manyinga waze akuneha chindululu (glucose) ha manyinga ha mukunda wa uhenge wa insulin, hanji ha yuma yeswe yivali.
Pathophysiology ya yikola ya shuga yili ni yuma yinji ya ku mwono, ya ku musenge, ni yuma yikwo yize yakukwasa yikola yacho.
Ha mukunda wa yikola ya shuga ya type 1, yikola yacho yakukwata ha kulifumba cha ma celula beta akuneha insulina mu pancreas, chino chakuneha uhenge wa insulin.
Chino chakupwisa kukamba cha kulinga kanawa manyinga, ni kutwala ku hyperglycemia (kashika manyinga kupwa ni manyinga anji).
Ha mukunda wa yikola ya shuga ya 2 yikola yacho yili yikalu chinji nawa yakuneha chinyingi cha insulin ni kuhona cha insulin.
Pathophysiology ya yikola ya shuga yakukwasa nawa hanga yikumba yipema, ngwe kupinjisa manyinga, manyinga ni yihanda, yize muyihasa kutwala ku yikola yinene ya mbunge, yikola ya mafwa, yikola ya mafwa ni kupomba cha meso.
Kunyingika kanawa yikola ya musongo wa shuga chili chilemu chinji ha kukwasa atu hanga anyingike chize mahasa kuchiza ni kuchiza yitanga yacho.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
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Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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