La fisiopatologjie di un atac di cûr, cognossût ancje come infarto miocardic, e compuarte la interuzion dal flus di sanc viers une part dal muscul dal cûr, che al puarte ae muart des celulis dal cûr.
Chest al sucêt par solit par vie de ostruzion di une arterie coronarie di un coagul di sanc, che spes al è il risultât di aterosclerosi, une cundizion dulà che la plache si costruìs intes arteris.
La plache e je fate di colesterôl, sostancis grasis, prodots di scart celulâr, calci e fibrine.
Cuant che une plache si rompe, e pues causâ la formazion di un coagul di sanc, che al pues blocâ la arterie e impedî al sanc siôr di ossigjen di rivâ al muscul dal cûr.
Cheste mancjance di ossigjen e cause la muart des celulis dal muscul dal cûr, puartant a un atac di cûr.
La entitât dal dam e dipent de grandece de aree furnide de arterie blocade e dal timp tra l'atac e il tratament.
I sintoms di un atac di cûr a puedin cjapâ dentri dolôr o fastidi tal pet, dispnea, nausea, sveniment e dolôr tai braçs, tai pîts, te massele o te schene.
Il tratament par un atac di cûr al impliche di solit il tornâ a meti il flus di sanc intal muscul dal cûr il plui ae svelte pussibil, cun lis medisinis o cun proceduris come la angioplastie e il stent o la operazion coronariche.
Al è impuartant notâ che la fisiopatologjie di un atac di cûr e je complesse e e cjape dentri diviers fatôrs, come gjenetics, stîl di vite e fatôrs ambientâi.
I fatôrs di risi pal atac dal cûr a includin la pression alte, il colesterôl alt, il fumâ, il diabete, la obesitât, la mancjance di ativitât fisiche e une storie di famee di malatie dal cûr.
Mantignî chescj fatôrs di risi al pues judâ a ridusi la probabilitât di vê un atac di cûr.
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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