Pathophysiology nke ọrịa shuga na-ezo aka n'ọmụmụ nke usoro anụ ahụ na-adịghị mma nke na-eduga n'ịzụlite ọrịa shuga.
Ọrịa shuga bụ ìgwè ọrịa metabolic nke a na-ahụ maka ọkwa shuga (glucose) dị elu n'ọbara nke na-esite na ntụpọ na secretion insulin, ọrụ insulin, ma ọ bụ ha abụọ.
Pathophysiology nke ọrịa shuga na-agụnye mmekọrịta dị mgbagwoju anya nke ihe ndị na-akpata mkpụrụ ndụ ihe nketa, gburugburu ebe obibi, na ụzọ ndụ nke na-enye aka n'ọrịa ahụ.
Na ọrịa shuga ụdị 1, pathophysiology na-agụnye mbibi autoimmune nke mkpụrụ ndụ beta na-emepụta insulin na pancreas, na-eduga na ụkọ nke mmepụta insulin.
Nke a na- eme ka a ghara inwe ike ịchịkwa ọkwa glucose n" ọbara , na- eduga n" ọrịa hyperglycemia (ọbara shuga dị elu).
Na ọrịa shuga nke ụdị 2, pathophysiology dị mgbagwoju anya ma na-agụnye ma insulin na-eguzogide ma na-emebi insulin secretion.
Insulin na-eguzogide ọgwụ na-eme mgbe mkpụrụ ndụ nke ahụ adịghị azaghachi insulin n'ụzọ kwesịrị ekwesị, na-eduga n'enweghị ike iji glucose eme ihe n'ụzọ dị irè maka ike.
Nke a na- eme ka pancreas mepụta insulin karịa iji gbalịa imeri nguzogide ahụ, mana ka oge na- aga, pancreas nwere ike ọ gaghị enwe ike ijide ọchịchọ ahụ, na- eduga na mbelata nke mmepụta insulin.
Ọrịa shuga nke ime, nke na-eme n'oge ime, na-esite ná mgbanwe hormone ndị na-eduga n'ịnagide insulin.
Placenta na-emepụta homonụ ndị pụrụ igbochi ọrụ nke insulin, na-eduga n'ịba ụba nke glucose n'ọbara.
Pathophysiology nke ọrịa shuga na-agụnyekwa mmepe nke nsogbu, dị ka mmebi nke akwara ọbara, akwara, na akụkụ ahụ, nke nwere ike ibute nsogbu ahụike siri ike dị ka ọrịa obi, ọrịa strok, ọrịa akụrụ, na ọnwụ ọhụụ.
Ịghọta pathophysiology nke ọrịa shuga dị oké mkpa maka ịzụlite ọgwụgwọ ndị dị irè na atụmatụ mgbochi maka ọrịa a na-adịghị ala ala.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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