Myit machyi ai lam a pathophysiology gaw hpa rai kun?
Myocardial infarction ngu ai myit masin ana a pathophysiology hta, masin salum daw mi de sai lwi ai lam hten za nna, masin salum a si ai lam byin ai.
Ndai gaw, sai htaw ai lam a majaw, sai lam yan pat ai majaw byin ai.
Dai hta cholesterol, sau, sai n'hkrung ni a jahten kau ai arai ni, calcium hte fibrin ni lawm ai.
Dai majaw sai htawng langai mi hten wa nna, sai htawng gaw sai lam hpe pat kau ya ai.
Ndai oxygen n law ai majaw, salum hta nga ai salum ni si mat nna, salum ana byin wa ai.
Dai machyi ai lam gaw, sai lam n shang ai shara hte ana byin ai aten ladaw hta madung rai nga ai.
Myit machyi ai lam a kumla ni gaw, sinda hta machyi ai, nbung loi ai, kan machyi ai, baw hten ai, lahpa, du, nra, shingma machyi ai lam ni re.
Myit machyi ai hpe tsi lajang ai lam hta, sai lam hpe lawan ai hku bai lu hkra galaw ra ai.
Dai hta n-ga, ana byin ai shaloi, ana kanu ni hte ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe mung, ana kanu ni hpe
Myit machyi ai lam byin wa hkra galaw ai lam ni gaw sai rawt ai, sai hta cholesterol law ai, tsa chyaru lu ai, sai htaw ai, hkum shan grai kaba ai, hkum hkrang n shamu shamawt ai hte nta masha ni hta myit machyi ai lam byin wa ai lam ni re.
Dai lam ni hpe atsawm sha galaw yang, myit machyi ai lam n byin ai.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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['hte (6) copyright madu a mying hte galaw na ahkang lu ai wa a hkum hkrang (sh) electronic signature.']
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['Matut Mahkai U']
['Gara hku mung san mayu yang email hte shana ya rit.']
What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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