Fisiopatolojia di diabeti ta rifiri a studu di prusésus fiziulójiku dizordenadu ki ta leba pa dizenvolvimentu di diabeti mellitus.
Diabéti mellitus é un grupu di duénsas metabóliku karaterizadu pa altu nível di asukar na sangi (glukózi) ki ta rezultadu di difétu na sekreton di insulina, na ason di insulina, ô na tudu dôs.
Fisiopatolojia di diabeti ta nvolve konpleksu interason di fatoris di genétika, meiu anbiental, y stilu di vida ki ta kontribui pa dizenvolvimentu di duénsa.
Na diabeti di tipu 1, patofisiolojia ta nvolve distruison autoimun di sélulas beta ki ta produzi insulina na pankréas, ki ta leba a un defisiênsia na produson di insulina.
Kel-li ta poi asukra na sangi ta bira más txeu.
Na diabétititi di tipu 2, patofisiolojia é más konplexu y ta nvolve tantu rezisténsia a insulina komu nun stadu di sékrison di insulina.
Rezisténsia a insulina ta kontise óras ki sélula ka ta reaji dretu a insulina.
Kel-li ta poi pankreas ta produzi más insulina pa tenta vense kel rezisténsia, má ku ténpu, pankreas pode ka konsigi faze más insulina ki el meste.
Diabéti gestacional, ki ta kontise duranti gravidês, é pamodi mudansa hormonal ki ta poi algen ta fika duenti.
Pur isu, sédu ô tardi, kel insulina ta bira más txeu na sangi.
Fisiopatolojia di diabeti tanbê ta envolvi dizenvolvimentu di konplikasons, sima danus na vasus di sangi, nervus y órgãus, ki podi leba a prublemas di saúdi gravi sima duénsa kardíaku, AVC, duénsa renal y pérda di vista.
Konprende patofisiolojia di diabeti é fundamental pa dizenvolve tratamentus i stratejias di prevenson efikas pa es duensa kroniku.
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Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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