Pathophysiologie ya cancer ya colon ezali kolobela ba mécanisme mpe ba processus oyo ememaka na bokono mpe bokóli ya cancer ya colon.
Cancer ya colon, eyebani mpe lokola cancer ya colorectal, ezali lolenge ya cancer oyo ebandaka na colon to rectum, oyo ezali biteni ya intestine monene.
Mbala mingi ebandaka na mwa eloko moko oyo ebimaka na nzoto, oyo babengi polypes, mpe nsima ya mwa ntango ekoki kokóma kanser.
Pathophysiologie ya cancer ya colon ezali na ba facteur ebele, na kati na yango ba mutation ya ba gène, inflammation, mpe ba facteur ya milieu.
Mbongwana ya ba gène ekoki kosalema na ADN ya ba cellules ya colon, oyo ekomema na bokóli ya ba cellules mpe bokabwani na yango.
Mbongwana yango ekoki kouta na makila to na maladi, mpe ekoki kobebisa ba gènes ndenge na ndenge oyo esalaka ete baselile ekola, ekabwana mpe emibongisa.
Kobima ya maladi na kati ya misɔpɔ, oyo ekoki kouta na maladi ya misɔpɔ, ekoki mpe kobakisa likama ya kozwa kanser ya misɔpɔ.
Soki kaka kanser ya colon ebimi, ekoki kokóma na biteni mingi, kobanda na ebandeli oyo ezalaka kaka na colon tii na biteni oyo eleki makasi, oyo kanser epalangani na biteni mosusu ya nzoto.
Baselile ya kanser ekoki kokɔta na binama ya nzoto oyo ezali pembeni, mpe ekoki mpe kopalangana na nzela ya makila tii na bisika mosusu, na ndakisa na libale to na mimpululu.
Kosalisa cancer ya colon esangisaka lipaso, chimiothérapie, mpe radiothérapie, na kotalela stade mpe esika ya cancer.
Koyeba maladi yango noki mpe kosalisa yango ezali na ntina mingi mpo na kobikisa yango, mpo mbala mingi maladi ya kanser ya estoma ekoki kobikisama soki bakangi yango noki.
Soki moto azali kosala ekzamɛ mbala na mbala, na ndakisa na nzela ya masini oyo esalisaka na komona misɔpɔ ya minene, yango ekoki kosalisa ye ayeba kanser ya misɔpɔ ya minene ntango ezali naino na ebandeli, ntango oyo ekoki kobika.
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Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
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What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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