La fisiopatologia del cancer del polmon la se riferiss ai cambiament ind i process e i mecanism fisiologich normal qe se verifican ind el desvilup e la progression del cancer del polmon.
El cancher de polmon l"è una malattia cumplessa che la nass de la cressita e divisiun incontrollada di cellule anormali ind i polmon.
Cheste cellule pœden formar tumors e sparger-s in olter part del corp, portand a vari sintom e complicazion.
La fisiopatologia del cancher de polmon la coinvolge vari fattur, includ i mutazion genetich, i fattur ambient e i scelte de stil de vita.
I mutazion genetich pœden capitar ind el DNA dei cellule polmonar, qe i porta a una cressida e division cellulare incontrolada.
Cheste mutaziun pœden vesser eredità o acquistade, e pœden vesser causade de l'esposiziun a sostanze cancerogene, come el fum del tabach, el radon, l'amiant e l'inquinament de l'aria.
El cancher de polmon el pœl vesser classificaa in du tip principai: el cancher de polmon a cellule picinine (SCLC) e el cancher de polmon a cellule no picinine (NSCLC).
La fisiopatologia de qesti tipi de cancher de polmon pœl vesser diversa, perqè g'han di mutazion genetich diferent e respond diferent al tratament.
La fisiopatologia del cancher del polmon la coinvolge anca l'interazion intra i cellule cancerose e el tessud circundant, compres el sistema immunitari.
I cellule cancerose pœden evità el sistema immunitari, permetend de cresser e de spantegar-se senza control.
Inoltra, el microambient del tumor pœl promœver la cressida del tumor e la metastasi fornind un ambient de supor per i cellule cancerose.
La fisiopatologia del cancher de polmon l"è un process cumpless e dinamic, e i ricercadur i lavoren de continuo per capì mej i mecanism che g'hinn dedree per sviluppà di tratament plussee efficaci e migliorar i resultad di pazient.
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Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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