Thinlung beihna (heart attack) chu thisen kalna a tihbuai avângin lung lam cell a thi ṭhîn.
Chu chu thisen pût avânga lungpui zungpui (coronary artery) a hnawh phui vâng a ni a, chu chu atherosclerosis, thisen zungpui chhûnga lungphu awm avânga lo awm ṭhîn a ni.
Chu lungphu chu cholesterol te, thau te, cell thil chhia te, calcium te, leh fibrin te siam a ni.
Chu chuan thisen zûk a siam thei a, chu chuan thisen zûk chu a dang chat thei a, chu chuan thisen oxygen ngah tak chu lungpuiah a thlen a dang chat thei a ni.
Chu oxygen tlâkchhamna chuan lung thazâm cell-te chu thihtîrin, chu chuan lungphu chawlhna a thlen a ni.
Chu chu a natna a nasat dân leh a natna vei leh enkawl a nih hun chhûng azirin a innghat a ni.
Thinlunga beih natna a lan chhuahna chu, pum na emaw, na lo emaw, thaw hah emaw, nausên emaw, luhai emaw, leh kut, hnar, hnuk na emaw a ni thei.
Thinlunga beih natna enkawl dân chu damdawi emaw, angioplasty leh stent emaw, coronary artery bypass surgery emaw hmanga a rang thei ang bera thinlunga thisen luang lût lehna a ni.
Heart attack natna (pathophysiology) chu a buaithlak hle a, genetic, lifestyle leh environmental factors te pawh a tel a ni.
Thinlung natna thlen theihna chhante chu thisen sâng, cholesterol sâng, meizial zu, diabetes, thau lutuk, taksa chak lo lutuk, leh chhûngkaw zîngah thinlunga natna vei an awm ṭhinte hi a ni.
Hêng thil hlauhawmte enkawlna chuan lung natna vei theihna a titlêm thei a ni.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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['(5) Thu dik lo sawi hlauvin i hming ziakna, hriattîrna thute chu a dikzia leh i bawhchhiat nia sawi dikna chanvo humhim tûra thuneihna i nei tih i puan chhuahna;']
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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