Patofisiologi kanker paru-paru marujuak pado parubahan pado proses jo mekanisme fisiologis normal nan tajadi dalam pakambangan jo parkambangan kanker paru-paru.
Kanser paru adolah panyakik kompleks nan muncua dari patumbuahan jo pambagian sel abnormal nan indak taatur di paru-paru.
Sel-sel iko dapek mambantuak tumor jo manyebar ka bagian tubuah lainnyo, nan manyababkan bamacam gejala jo komplikasi.
Patofisiologi kanker paruik maliputi babarapo faktor, tamasuak mutasi genetik, faktor lingkungan, jo pilihan gaya hiduik.
Mutasi genetik dapek tajadi pado DNA sel paruik, nan manyabaukan patumbuahan jo pambagian sel nan indak taatur.
Mutasi iko dapek diwariskan atau didapek, dan dapek disababkan dek paparan karsinogen, sarupo asok rokok, radon, asbes, jo polusi udaro.
kanker paru dapek diklasifikasikan manjadi duo jinih utamo: kanker paru sel ketek (SCLC) jo kanker paru sel indak ketek (NSCLC).
Patofisiologi dari tipe kanker paru-paru iko dapek babeda, karano inyo mampunyoi mutasi genetik nan babeda jo marespon babeda pado pangobatan.
Patofisiologi kanker paru-paru juo maliputi interaksi antaro sel kanker jo jaringan sakitarnyo, tamasuak sistem imun.
Sel kanker dapek malinduangi sistem kakebalan tubuah, sahinggo dapek tumbuah jo manyebar indak tatangkok.
Salain itu, lingkungan mikro tumor dapek maningkekan patumbuahan tumor jo metastasis jo manyadiokan lingkungan mandukuang untuak sel kanker.
Patofisiologi kanker paru adolah proses nan kompleks jo dinamis, jo panaliti taruih bakarajo untuak labiah mangarati mekanisme nan mandasarinyo untuak mangambangan pangobatan nan labiah efektif jo maningkekan hasil pasien.
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Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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