Naoor wʋsgo, yaa bũmb sẽn yit yĩngẽ wã n wat n lebg kãnsɛɛre.
Kãnsaarã bãas wata ne yɛl wʋsgo, wala makre, rog-n-mik toeengo, yĩns sãang la yɛl a taab sẽn kẽed ne vɩɩmã.
Zĩn-dãmbã tõe n wa kɩtame tɩ ninsaal yĩngã wils lebg wʋsgo, tɩ b pa le tõe n welg taab ye.
Zĩn-kãensã tõe n yaa rogem wall bũmb a to sẽn wat ne, la b tõe n toeema zɛlmã bɩʋʋngo, a welgre, la a manegre.
Kẽer me bẽeda kãnsɛɛrã.
Wẽnemã sẽn kaoosd wakat fãa wã tõe n kɩtame tɩ yĩngã wil yiisd bõn-yood sẽn kɩtd tɩ b bɩt n paasdẽ, la b welgdẽ, tɩ rẽ tõe n wa baas ne kãnsɛɛre.
Yɛl nins sẽn be tõnd vɩɩmã pʋgẽ wã, wala rɩɩbã, vɩɩmã manesem la kemikall kẽer tõe n kɩtame me tɩ kãnsɛɛr paam neda.
Wala makre, b wilgame tɩ ned sã n dɩt nemd sẽn yaa miuug wʋsg la a rɩt nemd b sẽn maneg sõma, tõe n kɩtame t'a bẽ kãnsɛɛrã.
Bũmb a taab me n tõe n kɩt tɩ bãagã paam-do. Wala makre, b sã n yaa bedr wʋsgo, b yũuda sɩgaare, la b pa maand yĩn-wɩsgr ye.
Kãnsaarã sã n wa yik n be, a tõe n kẽe yel-kãsems toor-toor pʋsẽ, sẽn sɩng ne a sɩngr wakatẽ wã, a sã n tɩ bee kãnsɛɛrã pʋgẽ, n tɩ tãag a sã n tɩ sãag yĩngã zĩis a taaba.
Bãasã tõe n kẽe yĩngã wil la a wil nins sẽn pẽ-bã pʋsẽ, la a tõe n yelga ne zɩɩmã bɩ ne zɩ-pellã n kẽng zĩ-zãrsẽ wala zɩ-biis wall zɩ-pɛla.
Bãas nins sẽn wat ne kãnsɛɛrã tɩɩm yaa woperasɩõ, kemoterapɩ, la radioterapɩ.
Bãaga sã n wa puk tɩ b pa reng n bãnge, la b sã n wa paam tɩ b tɩp-a, b tõe n sãooga bãaga.
B sã n maand wopeere wã vaeesg wakat fãa, tõe n sõngame tɩ b bãng kãnsɛɛrã sẽn sɩng to-to, tɩ b sã n wa yã-a bɩ b tõog n sãoog-a.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
['Bãngr-gomde: logtoeemdã']
['Yaa zãmsg la kibay kũun yĩng bal la b yiisd sit kãngã, la pa logtoeemdã wɛɛngẽ sagls bɩ tʋʋm-sõng maaneg yĩng ye.']
['B pa segd n tũnug ne kɩbay nins b sẽn kõ wã n bao bãag bɩ zu-loɛɛg tɩɩm ye. Sẽn dat-b tɩbsg b toor segd n tɩ yãa logtor sẽn tar sor n tõe n tɩp-ba.']
['D tẽeg tɩ b sẽn maand to-to n leokd sogsgã to-to wã pa zems ye.']
['Y sã n wa karemd bũmb sẽn be sɩtã pʋgẽ, bɩ y ra tol n yĩm tɩ y segd n tɩ gesa logtor ye. Y sã n tagsdẽ tɩ y tara yĩn-wɩsgr yell bɩ y bool 911 wall y kẽng logtor yirã.']
['Bas-m-yam: sɛbã zãab wɛɛngẽ']
['Digital Millennium Copyright Act sẽn yaa yʋʋmd 1998 soabã, 17 U.S.C. § 512 (DMCA) kõta sor tɩ ned sã n tẽed tɩ bũmb sẽn be ẽntɛrnetã pʋgẽ kɩɩsda a sẽn tar sor n tõe n maan bũmb ninsã, a tõe n tɩ kos n paam n lebse.']
['Yãmb sã n tẽed ne pʋ-peelem tɩ bũmb sẽn be tõnd sɩt wɛɛbã pʋgẽ bɩ tõnd tʋʋm-noyã pʋsẽ n sãamd yãmb dʋrwa wã, yãmb (bɩ y tʋʋm-tʋmdã) tõe n toola tõnd koɛɛg n kos tɩ d yiis bũmbã wall d gɩdg tɩ y ra paam n kẽ ye.']
['B segd n tʋma koees ne ẽtɛrnetã (Ges-y ẽtɛrnetã adɛrs sẽn be babg ning sẽn yet tɩ "Tõnd sõsg zĩigã").']
['DMCA wã baoodame tɩ yãmb sẽn na n togs ned tɩ b maan-a-la bũmb sẽn kɩɩsd a sẽn tar sor n tõe n maan bũmb ningã, bɩ y wilg-a bũmb nins sẽn pʋgdã: 1) bũmb ning sẽn kɩt tɩ b maan-a bũmb ningã, 2) bũmb ning sẽn kɩt tɩ b maan-a bũmb ningã, la y wilg-d bũmb ning sẽn kɩt tɩ d tõe n bãng a sẽn be zĩig ninga. 3) y sẽn tõe n paam ned n gom ne-a to-to, n paas y adɛrsã, telefõnnã nimero, la y e-mailã. 4) y sã n yeel tɩ y kɩsa sɩd tɩ bũmb ning sẽn kɩt tɩ y maan bũmb ningã pa ned ning sẽn tar sor n tõe n maan bũmbã, bɩ a tʋm-tʋmdã, bɩ laloa wã sẽn kõ sor tɩ y maan ye.']
['5) Y sã n wa rat n wilg tɩ y pa tar sor n na n kɩɩs ned a to, bɩ y gʋls sebr n wilg tɩ y sẽn togsã yaa sɩda, la tɩ y tara sor n na n wilg tɩ nedã sẽn maan bũmb ning n kɩɩs yãmb dʋrwa rãmbã yaa sɩda.']
['La (6) sɛb nins sẽn tar-b sor n na n yiis sɛbã, bɩ ned sẽn tar sor n na n tʋm sɛbã yiisg yĩngã.']
['Y sã n pa gʋls kibay nins sẽn be yĩngrã, tõe n kɩtame tɩ y yẽgengã kaoos n pa sa ye.']
['Sõsg ne neda']
['Y sã n tar sokr bɩ y sẽn dat n bãnge, bɩ y gʋls-d lɛtr n tool-do.']
What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
Disclaimer: medical
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Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
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Disclaimer: copyright
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['Sẽn zems']
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['D sɩngda ne sɛb milyõ 35 sẽn tik Biiblã zug n yiisd tɩbsg wɛɛngẽ.']
