Pathophysiology ea lefu la tsoekere e bolela thuto ea mekhoa e sa laoleheng ea'mele e lebisang ho hlaheng ha lefu la tsoekere.
Lefu la tsoekere ke sehlopha sa mafu a tšoaetsanoang ke lintho tse ling a tšoauoang ke tsoekere e ngata maling (glucose) e bakoang ke liphoso tsa ho ntša insulin, tšebetso ea insulin, kapa ka bobeli.
Pathophysiology ea lefu la tsoekere e akarelletsa ho sebelisana ho rarahaneng ha liphatsa tsa lefutso, tikoloho le mekhoa ea bophelo e tlatsetsang ho hlahiseng lefu lena.
Ho lefu la tsoekere la mofuta oa 1, pathophysiology e akarelletsa ho timetsoa ha lisele tsa beta tse hlahisang insulin ka har'a pancreas, e leng se lebisang ho haelloeng ha tlhahiso ea insulin.
Sena se fella ka ho se khone ho laola maemo a tsoekere maling, e lebisang ho hyperglycemia (tsoekere e phahameng maling).
Ho lefu la tsoekere la mofuta oa 2, pathophysiology e rarahane haholo'me e akarelletsa ho hanyetsa insulin le ho se sebetse hantle ha insulin.
Ho hanyetsa insulin ho etsahala ha lisele tsa'mele li sa arabele hantle ho insulin, e leng se etsang hore li se ke tsa khona ho sebelisa tsoekere hantle hore e be matla.
Sena se etsa hore manyeme a hlahise insulin e eketsehileng ho leka ho hlōla ho hanyetsa, empa ha nako e ntse e feta, manyeme a ka 'na a se ke a khona ho etsa seo a se hlokang, e leng se lebisang ho fokotseheng ha tlhahiso ea insulin.
Lefu la tsoekere la bokhachane, le hlahang nakong ea bokhachane, le bakoa ke liphetoho tsa lihormone tse etsang hore'mele o se ke oa khona ho sebelisa insulin.
Placenta e hlahisa lihormone tse ka thibelang tšebetso ea insulin, e leng se etsang hore tsoekere e maling e eketsehe.
Pathophysiology ea lefu la tsoekere e boetse e akarelletsa ho hlaha ha mathata, a kang ho senyeha ha methapo ea mali, methapo ea kutlo le litho, e ka lebisang mathateng a tebileng a bophelo bo botle a kang lefu la pelo, stroke, lefu la liphio le ho foufala.
Ho utloisisa pathophysiology ea lefu la tsoekere ke habohlokoa bakeng sa ho hlahisa mekhoa e sebetsang ea ho phekola le ho thibela lefu lena le sa foleng.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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