Pathophysiology ea lefu la pelo, le tsejoang hape e le myocardial infarction, e akarelletsa ho sitisoa ha phallo ea mali karolong ea mesifa ea pelo, e lebisang lefung la lisele tsa pelo.
Hangata sena se etsahala ka lebaka la ho thijoa ha tšilo ea pelo ke mali a koetsoeng, e leng se atisang ho bakoa ke lefu la ho thatafala ha methapo ea mali, e leng boemo boo ho lona ho bokellanang mali methapong.
Letlapa lena le entsoe ka k'holeseterole, mafura, litšila tse tsoang liseleng, calcium le fibrin.
Ha plaque e phatloha, e ka etsa hore ho be le mali a koahetsoeng, a ka thibelang tšelo ea mali'me a thibela mali a nang le oksijene hore a se ke a fihla mesifa ea pelo.
Ho haella hona ha oksijene ho etsa hore lisele tsa mesifa ea pelo li shoe, e leng se lebisang ho lefu la pelo.
Tšenyo e bakoang ke lefu lena e itšetlehile ka hore na tšebetso ea mali e etsoa sebakeng se seholo hakae le hore na lefu lena le tšoeroe nako e kae pele ho etsoa phekolo.
Matšoao a lefu la pelo a ka akarelletsa bohloko kapa ho se phutholohe sefubeng, ho hema ka thata, ho nyekeloa ke pelo, ho ba le hlooho e bobebe le bohloko matsohong, molaleng, mohlahareng kapa mokokotlong.
Hangata phekolo ea lefu la pelo e akarelletsa ho khutlisetsa mali mesifeng ea pelo kapele kamoo ho ka khonehang, ebang ke ka meriana kapa ka mekhoa e kang ho koala mali a methapo ea pelo le ho kenya stent kapa ho e potoloha.
Ke habohlokoa ho hlokomela hore pathophysiology ea lefu la pelo e rarahane'me e akarelletsa lintho tse ngata, ho akarelletsa liphatsa tsa lefutso, mokhoa oa bophelo le tikoloho.
Lintho tse ka bakang lefu la pelo ke khatello e phahameng ea mali, k'holeseterole e ngata, ho tsuba, lefu la tsoekere, botenya, ho se ikoetlise le hore lelapa le na le lefu la pelo.
Ho laola lintho tsena tse ka bakang lefu lena ho ka thusa ho fokotsa monyetla oa hore motho a tšoaroe ke lefu la pelo.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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