Pathophysiology kanker paru-paru ngarujuk kana parobahan dina prosés jeung mékanisme fisiologis normal anu lumangsung dina perkembangan jeung kamajuan kanker paru-paru.
Kangker paru nyaéta panyakit nu kompléks nu timbul tina tumuwuhna jeung ngabagi sél abnormal dina paru.
Sél-sél ieu bisa ngawangun tumor sarta sumebar ka bagian awak séjénna, ngabalukarkeun rupa-rupa gejala jeung komplikasi.
Pathofisiologi kanker paru-paru ngalibatkeun sababaraha faktor, kaasup mutasi genetik, faktor lingkungan, jeung pilihan gaya hirup.
Mutasi génétik bisa lumangsung dina DNA sél paru, ngakibatkeun tumuwuhna sél jeung ngabagi sél nu teu kakontrol.
Mutasi ieu bisa diwariskeun atawa diala, sarta bisa disababkeun ku kakeunaan karsinogen, saperti haseup bako, radon, asbés, jeung polusi udara.
Kanker paru bisa digolongkeun kana dua tipe utama: kanker paru sél leutik (SCLC) jeung kanker paru sél non-leutik (NSCLC).
Pathofisiologi jenis kanker paru ieu bisa béda, sabab boga mutasi genetik nu béda jeung ngaréspon béda kana pangobatan.
Fisiologi patofisiologi kanker paru ogé ngalibatkeun interaksi antara sél kanker jeung jaringan sabudeureunana, kaasup sistim imun.
Sél kanker bisa nyingkahan sistim imun, jadi bisa tumuwuh jeung sumebar tanpa kendali.
Sajaba ti éta, mikro lingkungan tumor bisa ngamajukeun tumuwuhna tumor jeung métastasis ku cara nyadiakeun lingkungan nu ngadukung sél kanker.
Pathofisiologi kanker paru nyaéta prosés anu kompléks jeung dinamis, sarta para panalungtik terus digawé pikeun leuwih ngarti mékanisme nu aya pikeun ngamekarkeun pangobatan nu leuwih éféktif jeung ngaronjatkeun hasil pasien.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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