Dɛn ne kokoram a ɛwɔ nsono mu no ho ade a ɛma obi nya yare no?
Kokoram a ɛwɔ nsono mu yare no kyerɛ nneɛma a ɛma kokoram no ba ne sɛnea ɛkɔ so no.
Kokoram a ɛwɔ nsono kɛse mu, a wɔsan frɛ no colorectal cancer, yɛ kokoram a efi ase wɔ nsono kɛse anaa nsono ketewa mu, a ɛyɛ nsono kɛse no fã bi.
Mpɛn pii no, efi ase sɛ biribi a ɛyɛ kɛse a wɔfrɛ no polyp, na bere rekɔ so no, ebetumi adan kokoram.
Nneɛma pii na ɛma obi nya kokoram, ebi ne awosu mu nsakrae, ahohyehye, ne nneɛma a atwa ne ho ahyia.
Awosu mu nsakrae betumi aba wɔ DNA a ɛwɔ nsono mu nkwammoaa mu no mu, na ama nkwammoaa no nyin na wɔakyekyɛ wɔn ho a wontumi nyɛ ho hwee.
Wobetumi anya saa nsakrae yi afi awo mu anaa obi na ama aba saa, na ebetumi aka awosu mu nneɛma ahorow a ɛma nkwammoaa nyin, kyekyɛ, na wosiesie wɔn ho no.
Sɛ obi nya nsamanwaw wɔ ne nsono mu a, ebetumi ama wanya nsamanwaw mu kokoram.
Sɛ obi nya yare a ɛmma ne ho ntɔ no a, ebetumi ama nkwammoaa no mu ayɛ den na asɛe, na ebetumi ama wanya kokoram.
Nneɛma a atwa yɛn ho ahyia te sɛ aduan a yedi, sɛnea yɛbɔ yɛn bra, ne nnuru bi a yɛde di dwuma nso betumi ama yɛanyare kokoram.
Sɛ nhwɛso no, wɔaka sɛ sɛ obi di nam kɔkɔɔ ne nam a wɔadi ho dwuma pii a, ebetumi ama wanya nsono mu kokoram.
Nneɛma foforo te sɛ kɛseyɛ mmoroso, sigaretnom, ne apɔw-mu-teɛteɛ a wonyɛ nso betumi ama woayare.
Sɛ kokoram nya yɛ obi a, etumi kɔ so yɛ no pii fi bere a ɛyɛ obi a ne kokoram no afi ase no kosi bere a ne kokoram no atrɛw akɔ nipadua no afã foforo.
Kokoram nkwammoaa no betumi afa nipadua no akwaa a ɛbɛn hɔ mu, na ebetumi afa mogya mu akɔ baabi a ɛwɔ akyirikyiri te sɛ berɛbo anaa ahurututu mu.
Sɛ obi nya kokoram a, nea wɔtaa yɛ ne sɛ wɔyɛ no oprehyɛn, nnuru a ano yɛ den a wɔde sa yare, ne nea ano yɛ den a wɔde sa yare.
Sɛ obi nya kokoram a, ɛsɛ sɛ otumi hu no ntɛm na ɔhwɛ no yiye.
Nhwehwɛmu a wɔyɛ daa te sɛ nea wɔfrɛ no colonoscopy no betumi aboa ma wɔahu kokoram a ɛwɔ nsono mu wɔ ne mfiase pɛɛ, bere a ɛyɛ mmerɛw sɛ wɔbɛsa no no.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
['Nsɛm a Ɛnsɛ sɛ Wɔka:']
['Wɔayɛ wɛbsaet yi sɛ wɔmfa nkyerɛkyerɛ na wɔmfa nkyerɛkyerɛ afoforo, na ɛnyɛ sɛ wɔde rema aduruyɛ ho afotu anaa wɔde rema adwuma.']
['Ɛnsɛ sɛ wɔde nsɛm a wɔde ama no di dwuma de hwehwɛ yare bi ho yare anaa wɔde sa yare, na ɛsɛ sɛ wɔn a wɔrehwehwɛ ayaresa ho afotu no kohu oduruyɛfo a ɔwɔ tumi krataa.']
['Yɛsrɛ wo hyɛ no nsow sɛ, sɛnea wɔhwɛ nsɛmmisa no so no, sɛ ɛba sɛ wɔrekyerɛw nnipa dodow a wɔanya yare bi ho asɛm a, ɛntaa nsi yiye.']
['Hwehwɛ afotu fi wo duruyɛfo anaa ɔyaresafo foforo a ɔfata hɔ bere biara wɔ yare bi ho. Nnya adwene sɛ wo nsa bɛka oduruyɛfo afotu anaasɛ wubetwa so esiane biribi a woakenkan wɔ wɛbsaet yi so nti. Sɛ wususuw sɛ ebia wo ho behia wo wɔ ayaresa mu a, frɛ 911 anaa kɔ ayaresabea a ɛbɛn wo paa ntɛm ara.']
['Nsɛm a wɔmmɔ ho ban:']
['Digital Millennium Copyright Act a wɔhyɛe wɔ afe 1998 mu, 17 U.S.C. § 512 (DMCA) no ma wɔn a wɔwɔ hokwan sɛ wɔyɛ wɔn nneɛma no kwan sɛ wɔyɛ nea wɔpɛ biara.']
["Sɛ wugye di sɛ nsɛm anaa nneɛma a ɛwɔ yɛn wɛbsaet anaa yɛn dwumadibea no mu bi to wo mmara a wode bɔɔ nneɛma ho ban no a, wo (anaa w'ananmusifo) betumi de krataa akɔma yɛn de aka sɛ yɛnyi nsɛm anaa nneɛma no, anaa yɛmmɔ kwan mma wonnya bi."]
["Ɛsɛ sɛ wɔde krataa ne email na ɛbɔ amanneɛ (hwɛ 'Contact' afã hɔ na wubehu email address)."]
['DMCA hwehwɛ sɛ wo amanneɛbɔ a ɛfa nea wɔkyerɛ sɛ ɛyɛ mmara a wobu so ho no de nsɛm a edidi so yi ka ho: (1) adwuma a mmara bɔ ho ban a wɔkyerɛ sɛ wɔadi so no ho asɛm; (2) nsɛm a wɔkyerɛ sɛ wɔadi so no ho asɛm ne nsɛm a ɛbɛboa yɛn ma yɛahu baabi a ɛwɔ; (3) wo ho nsɛm a yɛde bedi nkitaho, a wo address, telefon nɔma ne email address ka ho; (4) wo nsɛm a ɛkyerɛ sɛ wugye di sɛ nea wɔabɔ ho sobo no nyɛ nea mmara ma ho kwan; ']
['(5) wo nsahyɛ a ɛkyerɛ sɛ nsɛm a ɛwɔ amanneɛbɔ no mu yɛ nokware, na wowɔ tumi sɛ wode nea wɔkyerɛ sɛ woadi ho dwuma no bedi dwuma; ']
['ne (6) nea ɔwɔ tumi sɛ ɔyɛ biribi ma obi a ɔwɔ tumi sɛ ɔyɛ biribi ma no no nsaano nkyerɛwee.']
['Sɛ woamfa nsɛm a yɛaka yi nyinaa anka ho a, ebetumi ama wo ka no akyɛ.']
['Nkitahodi']
['Yɛsrɛ wo, fa nsɛmmisa anaa nyansahyɛ biara a wowɔ brɛ yɛn wɔ e-mail so.']
What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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['Ɛfa ho']
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