糖尿病的病理生理學是指研究導致糖尿病發展的生理過程.
糖尿病是一群代謝性疾病, 症狀為高血糖 (葡萄糖) 水平,
糖尿病的病理生理學涉及遺傳,環境及生活方式因素的複雜相互作用,
胰腺內產胰島素的β細胞會被自身免疫系統破壞,
這會導致血糖水平無法調節,
糖尿病患者必須在使用藥物後,
胰島素抵抗是指身體細胞對胰島素反應不佳,
胰腺產生更多胰島素以克服抗藥性, 但隨著時間的推移,
孕婦糖尿病是因為荷爾蒙的改變而導致胰島素抵抗.
胎盤產生能阻擋胰島素作用的激素,
糖尿病的病理生理學也涉及合併症的發展, 例如血管,神經和器官的損傷,
了解糖尿病的病理生理學對於開發有效的治療和預防策略至關重要.
PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html
RefinedWeb https://arxiv.org/abs/2306.01116
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Palicka V: Pathophysiology of Diabetes Mellitus. EJIFCC. 2002, 13 (5): 140-144.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
['提供醫療建議或專業服務.']
['提供給我們的資訊不應用於診斷或治療健康問題或疾病,']
['特別是數字內容不準確, 例如確診患有特定疾病的人數.']
['請隨時向您的醫生或其他合格的醫療人員諮詢您的病情. 請勿因為您在本網站上閱讀的內容而忽視專業醫療建議或延遲尋求. 如果您認為您可能有醫療緊急情況,請立即致電911或前往最近的急診室. 本網站或其使用並未建立醫師-病人關係. BioMedLib及其員工或本網站的任何貢獻者,都不會就此提供的信息或其使用作出任何明示或暗示的聲明.']
['該法案為那些認為網路上出現的內容侵犯了他們在美國版權法下的權利的版權所有者提供了補救. ']
['您 (或您的代理人) 可向我們發送通知, 要求移除內容或資料,或封鎖存取.']
['請見"聯絡方式"一節.']
['要求您的侵權聲明包含以下資訊: (1) 侵權聲明所涉及的著作的描述; (2) 侵權聲明所涉及的內容的描述及足以讓我們找到內容的資訊; (3) 您的聯絡方式,']
['(5) 您在作假宣誓的懲罰下簽署的聲明, 表示通知中的資訊是正確的,']
['且 (6) 版權所有者或授權代表其行動的人士的實物或電子簽名.']
['請填寫您的投訴內容.']
['請發送電子郵件給我們,']
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
This web site is provided for educational and informational purposes only and does not constitute providing medical advice or professional services.
The information provided should not be used for diagnosing or treating a health problem or disease, and those seeking personal medical advice should consult with a licensed physician.
Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
Always seek the advice of your doctor or other qualified health provider regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website. If you think you may have a medical emergency, call 911 or go to the nearest emergency room immediately. No physician-patient relationship is created by this web site or its use. Neither BioMedLib nor its employees, nor any contributor to this web site, makes any representations, express or implied, with respect to the information provided herein or to its use.
The Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the “DMCA”) provides recourse for copyright owners who believe that material appearing on the Internet infringes their rights under U.S. copyright law. If you believe in good faith that any content or material made available in connection with our website or services infringes your copyright, you (or your agent) may send us a notice requesting that the content or material be removed, or access to it blocked. Notices must be sent in writing by email (see 'Contact' section for email address) . The DMCA requires that your notice of alleged copyright infringement include the following information: (1) description of the copyrighted work that is the subject of claimed infringement; (2) description of the alleged infringing content and information sufficient to permit us to locate the content; (3) contact information for you, including your address, telephone number and email address; (4) a statement by you that you have a good faith belief that the content in the manner complained of is not authorized by the copyright owner, or its agent, or by the operation of any law; (5) a statement by you, signed under penalty of perjury, that the information in the notification is accurate and that you have the authority to enforce the copyrights that are claimed to be infringed; and (6) a physical or electronic signature of the copyright owner or a person authorized to act on the copyright owner’s behalf. Failure to include all of the above information may result in the delay of the processing of your complaint.
['生物醫學圖書館使用自動化計算機 (機器學習算法) 來生成問答對.']
['開始於3500萬份的生物醫學出版物.']
['請見"參考資料"以及"聲明".']