Pathophysiology ya kuhwima cha mbunge, yize anavuluka nawa ngwo myocardial infarction, yakuneha kuhwima cha manyinga mu kachi ka mbunge, ni kutwala ku kufwa cha ma celula ja mbunge.
Chino chili kulingiwa ha mukunda wa kukoka cha manyinga ha mukolo ha mukolo, chino chili kulingiwa ha mukunda wa yikola ya arteries, yipikalo yize yakukoka manyinga ha mukolo.
Chikuto chacho chili ni cholesterol, mafwo, yisushi, calcium ni fibrin.
Muze plaque yakuputuka, muchisa manyinga hanga aputuke, chino chakukinda manyinga hanga achine kukwata manyinga waze ali ni oksijene hanga ahete ku mbunge.
Chino chakuneha yikola ya mbunge.
Ulemu wa yikola yacho unatale ha chize manyinga akuchikuka ni ha shimbu lize akupalika mba akuhwise.
Yisoneko ja kapinda kakukwata ku mbunge jinanyingika ngwe kuli yikola mu mbunge, kuhwima chikweta, kuhomba, kuputuka kukwata ni yikola ku moko, ku mbunga, ku mahuma hanji kunyima.
Uhashi wa kuchiza yikola ya mbunge unalumbunuka kufumisa manyinga hanga ahete kanawa ku mbunge, ni uhashi wacho kakuulinga nichimwe yitumbo hanji maukwaso ngwe angioplasty ni stenting.
Chili chilemu kunyingika ngwo pathophysiology ya kuhwima cha mbunge yili yikalu chinji ni kufupiwa ni yuma yinji, ngwe yuma ya ku mwono, ni yuma ya mu chifuchi.
Yuma yize yakuneha yikola ya mbunge yili: manyinga anji, cholesterol yinji, kunwa makanya, yikola ya shuga, kukola chinji, kuhona kulinga yuma yize yakukolesa mujimba, ni yikola ya mbunge yize akupwa nayo mu usoko.
Kukaula yitanga yacho muchihasa kukwasa kukehesako yikola ya mbunge.
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Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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