Pathophysiology ya cancer ya mahinji yinalumbunuka kwalumuka cha yitanga ni ndako ja mahinji yize yakulingiwa ha kukola ni kukola cha cancer ya mahinji.
Kanser ya yipapu yili yikola yize yakwiza ha kukola ni kulihandununa cha ma celula waze keshi ni uhashi wa kulifunga mu yipapu.
Maselusi jacho mahasa kuputuka kupwa ni yikumba ni kuhanda ni kulifumba mu yihanda yikwo ya mujimba, ni kuneha yikola yinji.
Pathophysiology ya cancer ya mahinji yakukwata ha yuma yinji, ngwe kwalumuka cha cha mujimba, yuma ya mu chifuchi, ni chize atu akutwama.
Mutações genéticas mahasa kulingiwa ha DNA ya célula ya pwapwa, ni kutwala ku kukola ni kulihandununa cha célula.
Mutação jacho kakujitambula kuli ana hanji kuli mutu mwene, nawa kakujihisa ha mukunda wa yikola ya kansa, ngwe makanya, asbestos, ni yuma yize yakuneha yikola yacho.
Kanser ya fupa kakuyihandununa mu mahanda aali: small cell lung cancer (SCLC) ni non-small cell lung cancer (NSCLC). NSCLC kakuyihandununa nawa mu mahanda atatu: adenocarcinoma, squamous cell carcinoma, ni large cell carcinoma.
Pathophysiology ya yitanga yino ya cancer ya mahinji yakulisa, mumu ayo kakwete kapinda munji wa mutagen ni kukumbulula chisa ha yitanga.
Pathophysiology ya cancer ya mahinji yakukwata nawa ha kulivwashana cha ma celula a cancer ni yikuma yize yakundama, kuhakilako ni sistema ya mujimba.
Maselusi a cancer mahasa kukumba tachi ja mujimba, chino chakukwasa hanga akole ni kulianga chakuhona kwaanyingika.
Nawa, yihela yize yili ni yikola yacho muyihasa kukwasa yikola yacho hanga yijihe hanji yifuke.
Pathophysiology ya cancer ya mahinji yili yuma yize yikwete ulemu unji, nawa akwa-kuhengula mashimbu eswe kakusa tachi hanga anyingike kanawa yuma yize yakuneha yikola yacho hanga ahase kuwana yitumbo yipema ni kuhwisa yikola yacho.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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