Pathophysiology nke nkụchi obi, nke a makwaara dị ka myocardial infarction, na-agụnye nkwụsị nke ọbara na-aga n'akụkụ nke akwara obi, na-eduga n'ọnwụ nke mkpụrụ ndụ obi.
Nke a na-emekarị n'ihi mgbochi nke akwara akwara site na mkpụkọ ọbara, nke na-abụkarị nsonaazụ nke atherosclerosis, ọnọdụ ebe ihe mgbochi na-etolite na akwara.
Ihe mejupụtara ụrọ ahụ bụ cholesterol, abụba, ihe ndị na-esi n'ahụ́ mkpụrụ ndụ apụta, calcium, na fibrin.
Mgbe ụrọ ahụ gbawara, ọ pụrụ ime ka ọbara kpọnwụọ, nke pụrụ igbochi akwara ahụ ma gbochie ọbara jupụtara na oxygen iru akwara obi.
Enweghị oxygen a na-eme ka mkpụrụ ndụ akwara obi na-anwụ, na-eduga n'ọrịa obi.
Otú ihe ahụ merụrụ ahụ́ hà na-adabere n'otú akwara ahụ e mechiri emechi hà, nakwa n'oge dị n'agbata mgbe ọrịa ahụ malitere na mgbe a gwọrọ ya.
Mgbaàmà nke nkụchi obi pụrụ ịgụnye ihe mgbu n'obi ma ọ bụ ahụ erughị ala, iku ume ngwa ngwa, ọgbụgbọ, isi ọwụwa, na ihe mgbu n'aka, n'olu, n'egedege ihu, ma ọ bụ n'azụ.
Ọgwụgwọ maka nkụchi obi na-agụnyekarị iweghachi ọbara na-aga n'akwara obi ngwa ngwa o kwere mee, ma ọ bụ site n'ịṅụ ọgwụ ma ọ bụ usoro ndị dị ka angioplasty na stenting ma ọ bụ ịwa ahụ akwara coronary.
Ọ dị mkpa iburu n'uche na pathophysiology nke nkụchi obi dị mgbagwoju anya ma na-agụnye ọtụtụ ihe, gụnyere mkpụrụ ndụ ihe nketa, ụzọ ndụ, na ihe ndị metụtara gburugburu ebe obibi.
Ihe ndị na-akpata ọrịa obi na-agụnye ọbara mgbali elu, cholesterol dị elu, ịṅụ sịga, ọrịa shuga, ibu oké ibu, ịghara ịna-emega ahụ́, na inwe ọrịa obi n'ezinụlọ.
Ịchịkwa ihe ndị a na- akpata ihe ize ndụ pụrụ inye aka belata ohere nke ịrịa nkụchi obi .
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LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
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Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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