Ti pathophysiology ti atake ti puso, a maawagan met iti myocardial infarction, ramanenna ti pannakasinga ti panagayus ti dara iti paset ti masel ti puso, a mangiturong iti ipapatay dagiti selula ti puso.
Kadawyanna a mapasamak daytoy gapu iti pannakabangen ti coronary artery babaen iti dara a timmangken, a masansan a resulta ti atherosclerosis, maysa a kasasaad a pakaigapuan ti panagurnong ti plaque kadagiti urat.
Ti plaque ket buklen ti cholesterol, taba, basura ti selula, calcium, ken fibrin.
No agsarut ti plaque, mabalin a tumaud ti maysa a dara a tumaud a mangserra iti urat ken manglapped iti dara a nabaknang iti oksihena a makadanon iti piskel ti puso.
Daytoy a kinakurang ti oksihena ti pakaigapuan ti ipapatay dagiti selula ti masel ti puso, a mangituggod iti atake ti puso.
Ti kadakkel ti pannakadangran agpannuray iti kadakkel ti lugar a serbian ti naserraan nga artery ken ti panawen manipud idi naatake agingga iti pannakaagas.
Dagiti sintoma ti atake ti puso ket mabalin a pakairamanan ti ut-ot iti barukong wenno di pannakaseknan, pannakabannog nga aganges, panagrikna iti nakaro a pannakaulaw, pannakaulaw, ken ut-ot kadagiti takiag, tengnged, pangal, wenno bukot.
Ti panangagas iti atake ti puso gagangay a ramanenna ti panangisubli iti dara iti masel ti puso iti kabiitan a panawen, babaen iti agas wenno pamay-an a kas iti angioplasty ken stenting wenno coronary artery bypass surgery.
Napateg a laglagipen a komplikado ti pathophysiology ti atake ti puso ken ramanenna ti adu a banag, agraman dagiti banag a mainaig iti gene, estilo ti panagbiag, ken aglawlaw.
Dagiti pakaigapuan ti pannakaatake ti puso ket pakairamanan ti nangato a presion ti dara, nangato a kolesterol, panagsigarilio, diabetes, kinalukmeg, kurang a pisikal nga aktibidad, ken adda kapamilia a nagsakit iti puso.
Ti panangtaming kadagitoy a mamagpeggad a banag ket makatulong a mangkissay iti posibilidad a maaddaan iti atake ti puso.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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