Patofisiologi kanker paru-paru nuduhake owah-owahan ing proses fisiologis normal lan mekanisme sing kedadeyan ing pangembangan lan perkembangan kanker paru-paru.
Kanker paru-paru minangka penyakit kompleks sing muncul amarga tuwuh lan pembagian sel abnormal ing paru-paru sing ora dikendhaleni.
Sel-sel kasebut bisa mbentuk tumor lan nyebar menyang bagean awak liyane, nyebabake macem-macem gejala lan komplikasi.
Patofisiologi kanker paru-paru kalebu pirang-pirang faktor, kalebu mutasi genetik, faktor lingkungan, lan pilihan gaya urip.
Mutasi genetik bisa kedadeyan ing DNA sel paru-paru, sing nyebabake tuwuh lan pembelahan sel sing ora dikendhaleni.
Mutasi kasebut bisa diwarisake utawa dipikolehi, lan bisa disebabake dening paparan karsinogen, kayata asap tembakau, radon, asbes, lan polusi udara.
Kanker paru-paru bisa diklasifikasikake dadi rong jinis utama: kanker paru-paru sel cilik (SCLC) lan kanker paru-paru non-sel cilik (NSCLC). NSCLC banjur dipérang dadi telung subtipe: adenokarcinoma, karsinoma sel skuamosa, lan karsinoma sel gedhe.
Patofisiologi jinis kanker paru-paru iki bisa beda-beda, amarga duwe mutasi genetik sing beda-beda lan nanggapi perawatan kanthi beda.
Patofisiologi kanker paru-paru uga nglibatake interaksi antarane sel kanker lan jaringan sekitar, kalebu sistem kekebalan awak.
Sel-sel kanker bisa ngliwati sistem kekebalan, saéngga bisa tuwuh lan nyebar tanpa dikendhaleni.
Kajaba iku, mikro-lingkungan tumor bisa ningkatake tuwuhing tumor lan metastasis kanthi nyedhiyakake lingkungan sing ndhukung kanggo sel kanker.
Patofisiologi kanker paru-paru minangka proses sing kompleks lan dinamis, lan para peneliti terus-terusan ngupayakake luwih ngerti mekanisme sing ndasari kanggo ngembangake perawatan sing luwih efektif lan ningkatake asil pasien.
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Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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