Pathophysiologie ya crise cardiaque, eyebani mpe lokola infarctus ya myocarde, ezali na kati ya kobeba ya makila oyo ekendaka na eteni ya misisa ya motema, oyo ememaka na liwa ya baselile ya motema.
Yango esalemaka mingi mpo ete makila ekangami na misisa ya motema, oyo mbala mingi ezalaka mpo na maladi ya atherosclerose, maladi oyo esalaka ete plaque ekóma mingi na misisa.
Soki plaque epasuki, ekoki kosala ete makila ekangama, oyo ekoki kokanga nzela ya makila mpe kopekisa makila oyo ezali na oxygène ekóma tii na misisa ya motema.
Kozanga oxygène wana esalaka ete baselile ya misisa ya motema ekufa, mpe yango ememaka maladi ya motema.
Ndenge oyo maladi yango ekolaka etalelaka bonene ya esika oyo makila ezali koleka na nzela ya misisa oyo ekangami mpe ntango oyo elekaki kobanda ntango maladi yango ebandaki tii ntango basalisaki ye.
Bilembo ya maladi ya motema ekoki kozala mpasi na ntolo, mpema mokuse, koyoka libumu mpasi, motó mpasi, mpe mpasi na mabɔkɔ, na nkingo, na mbanga to na mokongo.
Mbala mingi, mpo na kosalisa maladi ya motema, esɛngaka kozongisa makila na misisa ya motema nokinoki, ezala na lisalisi ya bankisi to na lisalisi ya mayele mosusu, na ndakisa angioplastie to stenting.
Ezali na ntina koyeba ete pathophysiologie ya crise cardiaque ezali na makambo mingi mpe esangisi makambo mingi, lokola ba facteurs génétiques, ya bomoi mpe ya milieu.
Makambo oyo ekoki kosala ete moto azwa maladi ya motema ezali tansiɔ, cholestérol, komɛla makaya, diabɛti, monene, kozanga kosala ngalasisi, mpe soki moto moko ya libota na yo abɛlaka maladi ya motema.
Soki ozali koyeba makambo oyo ekoki kosala ete obɛla maladi ya motema, yango ekoki kosalisa yo okima yango.
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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