Kodi pathophysiology ya khansa ya m'mapapo ndi yotani?
Pathophysiology ya khansa ya m'mapapo imatanthauza kusintha kwa njira za thupi ndi njira zomwe zimachitika pakukula ndi kupita patsogolo kwa khansa ya m'mapapo.
Khansa ya m'mapapo ndi matenda ovuta kwambiri amene amayamba chifukwa cha kukula ndi kugaŵanika kosalamulirika kwa maselo a m'mapapo.
Maselo ameneŵa angapangitse zotupa ndi kufalikira ku mbali zina za thupi, kutsogolera ku zizindikiro ndi mavuto osiyanasiyana.
Matenda a khansa ya m'mapapo amaphatikizapo zinthu zingapo, kuphatikizapo kusintha kwa majini, zinthu za m'chilengedwe, ndi njira ya moyo.
Kusintha kwa majini kungachitike mu DNA ya maselo a m'mapapo, kuchititsa kukula kwa maselo ndi kugaŵikana kosalamulirika.
Kusintha kumeneku kungakhale kwa choloŵa kapena kupezeka, ndipo kungayambitsidwe ndi zinthu zimene zimayambitsa kansa, monga utsi wa fodya, radon, asbestos, ndi kuipitsa mpweya.
Khansa ya m'mapapo imatha kugawidwa m'mitundu iwiri: khansa ya m'mapapo ya maselo ang'onoang'ono (SCLC) ndi khansa ya m'mapapo ya maselo osakhala ang'onoang'ono (NSCLC). NSCLC imagawidwa m'magulu atatu: adenocarcinoma, squamous cell carcinoma, ndi large cell carcinoma.
Matenda a khansa ya m'mapapo a mitundu imeneyi angakhale osiyana, chifukwa ali ndi kusintha kwa majini kosiyana ndipo amayankha mosiyana pa chithandizo.
Pathophysiology ya khansa ya m'mapapo imaphatikizaponso kuyanjana pakati pa maselo a khansa ndi minofu yozungulira, kuphatikizapo chitetezo cha m'thupi.
Maselo a khansa amatha kuthawa chitetezo cha m'thupi, n'kulola kuti akule ndi kufalikira popanda kuletsedwa.
Kuphatikiza apo, microenvironment ya chotupa imatha kulimbikitsa kukula kwa chotupa ndi metastasis popereka malo othandizira maselo a khansa.
Pathophysiology ya khansa ya m'mapapo ndi njira yovuta ndi yosinthasintha, ndipo ofufuza akugwira ntchito mosalekeza kuti amvetsetse bwino njira zoyendetsera kuti apange mankhwala othandiza kwambiri ndikuwongolera zotsatira za odwala.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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Chonde titumizireni imelo ndi funso / lingaliro lililonse.
What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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